What about some of the prehospital research coming out about potentially delaying defib until SPO2 and ETCO2 targets are reached???
@farhanqadeer825 күн бұрын
haney what do you think about talk regarding post chest tube management , how to use tnk/ tpa dornase in large bore chest tube.... when to remove chest tube... how do you treat or manage air leak, persistent air leak, ... when do you consider eb valve ,,,, when to consider vats and transfer to facility for evaluation,,,, how to manage hemothorax and when to take tube out,,,,, how to manage empyema and when to consider tnk/dornase and how to manage it,,,,, post chest tube complication...i think chest tube placement is very simple procedure ...its the management and post chest tube care which is more important.. what do you think... do you think that you will like to do a session/ chat on it.
@brycep56746 күн бұрын
Temporarily engorging the vessel does nothing for the longevity of that vascular access. When the tourniquet is removed, the vessel will return to its smaller diameter, and now your catheter to vein ratio is not ideal. Utilizing ultrasound for vascular access and being able to KNOW the native size of the vessel (without a tourniquet)is a much more effective process.
@drews67707 күн бұрын
Does the Vascular Guy have any more KZbin content?
@sw61188 күн бұрын
What newer drugs are you comparing to? DOAC or warfarin?
@joeidoni81628 күн бұрын
Ok, in what way?
@passion555410 күн бұрын
I loved this talk about vascular access as a CVICU nurse and Vascular Access nurse. However, I do not believe that in the future low dose vasopressors will belong on any top of "floor" rather than a ICU. The logics is in nursing staffing ratios. Nurses in the ICU are usually capped at 2 patients (3 at most) but, nurses on the floor will usually carry a load for 4-6 patients. I do not believe that is a safe staffing ratio for consistent IV site monitors for close peripheral access monitoring. Great talk!
@RishabhB10 күн бұрын
Isotonic bicarb?
@abdoumsr777811 күн бұрын
Where would i found the whole lecture ?!
@daniellewestenmeyer844412 күн бұрын
We are good at recognizing delirium when it is beginning, but I feel like once you start to see the symptoms manifest, it’s too late. Like a train running off the rails. The text book things to do like open a window, reorient the patient, allow time for rest, never seem to work at that point. It starts to spiral from just forgetting the month then by 12AM they’ve had haldol, Ativan, and are in restraints which just makes everything worse. Then is just has to run its course. What are things you suggest to deal with delirium once it starts?
@irynaYe13 күн бұрын
Wow....thank you!!!
@positronisomer20614 күн бұрын
“Chest compressions are good”
@ACCJohnMane14 күн бұрын
I would usually go for: - Etomidate - Rocuronium - Any Anaesthetic e.g. Thiopentone
@ranjithkumar-rm8zw16 күн бұрын
Channel name of guy talking ?
@shimonfriedman179715 күн бұрын
Rishi Kumar MD
@Maleksikder-p2d16 күн бұрын
Garcia Donna Perez Eric Lee Timothy
@sasha-taylor16 күн бұрын
I love seeing this procedure done, it's fascinating how well it can work Me encanta ver este procedimiento realizado, es fascinante lo bien que puede funcionar.
@yungceejay619216 күн бұрын
I would only ever use saline if i were stuck on a deserted island and had to make my own iv fluid.
@danymancuri16 күн бұрын
what about the concept of ZEEP (0 PEEP)? any comments on that? Also, better place the trigger "off" or as low as possible.
@ShadyRamirez-g1s17 күн бұрын
Thanks Edwin but it doesn't work good with my methamphetamine and heart irregularities
@graveyarddoji962017 күн бұрын
My husband started magnesium and potassium after his one a fib event and has been fine since
@ياسرالوالي-م9ظ17 күн бұрын
not agree with you, Internal medicine is the first
@robertbowman632017 күн бұрын
Wouldn't change working in the emergency department. The shift work and flipping days and nights definitely wears on you though.
@patc176417 күн бұрын
Had to listen again because music was distracting
@alialahmari588817 күн бұрын
please talk about the following, 1. The maximum doses in peripheral vs central line of KCL, 2. The maximum dose KCL per Hr if I have multiple central lines and the pt in severe hypoK. 3. KCL dose in cardiac arrest
@tareksasa94018 күн бұрын
Source, PMID plz!
@joshmcgoo19 күн бұрын
or you could just use an ultrasound
@scarzu972816 күн бұрын
Facts, if available.
@jeremymanley788219 күн бұрын
I tried this after it was posted on your insta page. Works well. Found it works great on geriatrics.
@4238945920 күн бұрын
Pneumo with needle decompression
@ianlondon288820 күн бұрын
How would abortion bands effect the care of this type of termination, assuming evacuation was the preferred intervention?
@thebearded_guardian367120 күн бұрын
Isn’t 600 mL VT a bit much intra arrest?
@romanishchenko631021 күн бұрын
Brilliant!
@amareswar260422 күн бұрын
Needle decompression of tension pneumothorax! Is there a bronchopleural fistula?
@thebearded_guardian367122 күн бұрын
Pneumothorax - looks like anterior needle thoracotomy
@joeidoni816222 күн бұрын
Every time I bring this up, I get poo-pooed because "AHA says..."
@mairossakorn635522 күн бұрын
Tracheobronchial tree injury
@SalmanAli-xd8kv22 күн бұрын
Needle decompression
@randys76622 күн бұрын
Everything is fine. Nothing to see here.
@theparaminuteman23 күн бұрын
Yikes
@davidpecora775023 күн бұрын
What exactly does “survival” rate mean? Definition used in this teaching brief. Thank you
@Reynosorex23 күн бұрын
Excellent!
@juliachambers72523 күн бұрын
Still don’t get why some doctors love dobutamine for heart failure and some would be noooo it’s gona cause arrhythmia and than we have to start lidocaine etc? I have cv docs prefer milrinone cuz it’s more awesome for right side and reduces pulmonary and pvr pressures. Some do both? So which one is better in which situation?
@tareksasa94023 күн бұрын
Plz insert the references for further reading
@donyzac24 күн бұрын
Enjoyed your lecture Alice!
@thebearded_guardian367124 күн бұрын
I understand in the super sick, they have increased insulin resistance which increases lipase production and potentially causing an acidosis. Similarly the increased cortisol levels of the super sick synergistically increases lipase. My question is, if this is true, wouldn’t vasopressin exacerbate that and increase the risk of developing a lactic acidosis or maybe keto acidosis?
@rumit994625 күн бұрын
❤it
@alptekinakturk418525 күн бұрын
best summary ever
@AnjaliSharma-sq2dy25 күн бұрын
But AHA Guidelines say give every 3 to 5 mins..and there is no upper limit! So plz guide