Studying or my CRNA school interview and this was very helpful! Appreciate the content!
@michellejohnston62603 жыл бұрын
I am watching every single one of your videos and they are helping me tremendously review for my board exam! Thank you 🙏🏻
@nygeek64712 жыл бұрын
The bradycardia isn’t from alpha 2 agonism.
@BarrelCollective4 ай бұрын
Is the bradycardia caused by baroreceptor activation/HTN that maintains with higher doses?
@hasitagoda39894 жыл бұрын
Can it be used in paediatric patients for day care procedures or to supplement during regional techniques
@RyanBeneby4 жыл бұрын
Thank you.
@corneliacarden43443 жыл бұрын
Thank you
@MennaMohamed-zb4hq Жыл бұрын
very good . thank you👏
@muhammadjalil44314 жыл бұрын
Nice, thanks
@dsrahul95113 жыл бұрын
Beautiful
@johnolive94773 жыл бұрын
Awesome!
@samuelcarlsen14423 жыл бұрын
I don’t really understand if this could theoretically be used as an actual general anesthetic? I know that it’s not used as such yet but I’m just curious to know if it theoretically could be possible? or if it’s too weak to produce Anesthesia or something else contraindicates this use?
@pacificprospector9 ай бұрын
I really wish vet would give more "full disclosure" regarding their use of buprenorphine with dex-medetomidine for short-term "sedation"....they said they would administer a reversing agent (for the Presidex) but the buprenorphine was not reversed (and really can't be) and b/c of the 22 hour half-life...it took our dog around 4-5 days to recover from a simple X-ray procedure. This is the danger when drug companies start pushing drugs to vets, who themselves don't really understand just what they are handling. I guess Vets don't want to handle the short-acting narcotics these days. Our other dogs recovered within hours after these kinds of procedures in the past. So the medetomidine was "controllable" but the buprenorphine they use in combo screwed things up. Ask questions people when you go to the vet and get clear answers.