Effect of Exercise and Cancer on Glycolysis

[@hamedhosseini4938]

Hi Andrey, just here to congratulate you because of reaching100k subs which is great achievement, I'm so happy for you. Although you deserve mills. You saved me throughout out my first year uni biology. All the best to you!

[@AKLECTURES]

Thank you for your kind words! I appreciate that! and thanks for watching my content! :)

[@erinbraun9171]

Your lectures are so great.

[@MatthewRawlik]

spell check: 3-phosphoglycerate becomes 2-phosphoglycerate in step 8 lol i have too much time on my hands

[@MardinUzeri]

Great lecture!

[@violinist86]

Very good break down of the pathways in the two circumstances (exercise and cancer) So in the case of cancer, how do you increase the inhibition of the HIF-1 so VEGF is not stimulated? Make the environment more alkaline to change the pH? Would exercise be contraindicated for a cancer pt?

[@AKLECTURES]

Isaac Murphy Thanks! Actually, drugs are currently being developed (perhaps already have been developed, not sure) that will target the HIF-1 molecules of cancer cells. The hope is to slow down blood vessel growth and cut off cancer cells' ability to form ATP.

[@moisesguel7132]

I fucking love you, man. Thanks, thanks, I really appreciate these lectures. The way you explain them is really really awesome. I think you should make a better publicity of your lectures, cause they are really good. People don´t really know about them, but if they knew it, they would love it same way as I do. Thanks, hopefully you can keep uploading such as awesome information.

[@MatthewRawlik]

HIF-1 also has an effect on Lactate Dehydrogenase

[@junczhang]

thanks for the lecture!

[@kchong228]

Thanks for ur lecture and the comparison between cells in exercises and cancer cells development. Eventually, do u mean we should not sprint but keep slow aerobic exercises only to prevent from cancer cells developing? I’m wondering how to apply this knowledge.

[@alaa2581]

please make vedioes about cancer

[@backstreetfan2887]

He said that NAD+ gets regenerated in the citric acid cycle, but shouldn't that be NAD+ gets regenerated in the electron transport chain?

[@mohamedibrahim-ds5xr]

after the cancer cells activate the hypoxade condition and vgef and have new blood supply they use the glucose . i have two question first >>>in the cancer cells condition is it fermentation condition stop when there is oxygen in the newly blood vessels ? second >>in the muscle cells after the new blood vessels come in the tired muscle cells they come back normal because of the glucose or because of the oxygen supply ?

[@CitraTheKrumZ]

thank your for the lecture, what book/source did you use in case of the HIF-1 mechanism? thanks

[@temitopeadeyemi7393]

Hi! thank you for the video, I was counting the enzymes and realized that phosphoglucooisomerase was missing, is there a reason for this?

[@beyzademirtas9768]

please turkish subtitles