Salmonella pathogenesis

[@GRHM310]

Few quick points: lipopolysaccharide is not secreted: it is a major component of the gram-negative envelope. Rather, this endotoxin is released either through bacterial lysis or as a result of bacterial replication . Additionally, endotoxin and LPS aren't technically the same thing; endotoxin is LPS + associated lipids from the bacterial membrane. Lastly, the O-antigen is not necessarily antigenic; it depends on the composition of the O-polysaccharide. Rather, it is the lipid A portion of the lipopolysaccharide that is the most immunogenic part of the molecule.

[@chintuvemula99324]

Amazing explaination. Brother ❤️

[@shomusbiologyofficial]

Thank you

[@thajulhidaya3009]

White boards are the best way... better use it sir...explanations are really good...

[@shomusbiologyofficial]

Yes.

[@thismoment9897]

here is a little little request. the way you teach is amazing..i love it.thats why i subscribed you sir.! . please sir if you are able to teach us with a whiteboard with your expressions,that is the best. or if you are unable for it ,please draw the pictures for each and every special words,incidents .these videos will get more and more clear then.thank you.😊😊😊.you are the super duper teacher in science in this you tube platform...

[@jahanzaibsaleem1995]

It means s.typhi doesn't enter blood stream??

[@aminudanjumaliadi9643]

It does

[@pauline836]

Do I need to include pathogenesis in explaining pathiphysiology for a case study to be presented in school?

[@khawajadilawarahmed7096]

Can anyone please tell, which software is this?

[@shomusbiologyofficial]

Paint

[@mrhb9033]

Any link?

[@drshahnafazal451]

What about payers patches

[@manraazz]

thanks for the great explanation..

[@mriduchaudhary9111]

Thanks

[@abdulrahaman0157]

Super bro❤️

[@shomusbiologyofficial]

Thank you

[@melibea13]

Greatly explained, thanks! I just have a couple of doubts left, please could you help me out? This mechanism of pathogenesis is the one typically followed by Salmonella Typhi, right? As far as I understand, the mechanism of action of typical agents of non typhoid salmonellosis, such as S. Enteritidis, consists simply on their invasiveness of the enteric epithelium and subsequent inflammation of the gut with water loss, sepsis being a complication by some subspecies. Am I right? Also, I have read somewhere that Salmonella Typhi, as opposed to the rest of Salmonellas, produces enterotoxins, but I don't see how that's relevant to their pathogenesis or how that fits in the process you've just described. I would be hugely grateful if you could clear this up for me!! Cheers! - a med student in Spain.

[@mafarmerga]

I know it would be a lot more work but if you could close caption this video then I could use it in my classes.

[@Thrishala100]

this was helpful, thnk u!

[@shomusbiologyofficial]

You're welcome

[@hruthvikbojja9459]

i like your videos but can you please make your videos more clear

[@lokeshdahal1374]

absolute rubbish,no mentioning of m cells,peyers patches

[@studypedia6698]

Thank you saved my 12 13 minta may be the explaination is not for clg students

[@humbertoluebbert7968]

I want to know if it enters into human cells

[@amjadzubair8421]

exactly the salmonella is facultative intracellular bacteria so need to invade the cells to save it live.

[@abdelatifsaou6981]

thank you

[@sampannamalla6750]

this had to be a 6 minute video if you didnt rkeep repeating what you were saying.

[@hehehaha8224]

Major problem with this guy 😂

[@MukeshKumar-cy5uc]

Not clear vedio sir we can not understand by this vedio

[@Chingkheinganba007]

neirehe

[@ashutoshmehra5455]

Sir apki video toh achhi hotii h ....but apka presentation achhi nhii h