Amazing explanation Doctor. Keep on making such legendry videos. Amazing Thankyou v much.

Good day ma'am. I used your videos for physiology and they were very helpful. Now I have started pharmacology and I want to use your videos now to first learn autonomic nervous system. Keep up the good work ma'am..... From Nigeria ❤

Ma'am my father suffered from hypertension....and BP around 170/100.. Aur doctor 3 tablets diye hai 1 din mei khane ko.... And now BP is within normal range.... But 3 hypertensive drug kya sahi hai???

It was the best on time, thankyou so much mam❤️🌹

Please mam, make concepts on final year topics ...exams are nearing 🥲

Tq for giving us awesome concept

3.35

Thank you maam🙌Very useful

Atlast i found it ❤

Dddddddd

Thankyou!!!

Thank you Madam.

Wow awesome ❤

Excellent explanation

My Mpv is drop 3.7 Bilirubin 1.1 borderline Creatine at borderline What test next mam . Pllz suggest correlation between these factors

Nice contents. Much understandable. Thank you madam

Mam any technique learn these drugs?

Mam please continue this series ...❤🙏🙏

Mam thanks please do continue this pharmacology teachings too, you helped me a lot in first year hope to do in second too

Another one😂 When bp goes low upto 80 mmhg , low MAP and thus low renal arterial pressure.. Thus reduce the rbf and reduce the gfr.. Thus less nacl in lumen of short segment of TAL of loop of henle lined by macula densa.. Thus less na cl move inside the mdc via nkcc2.. Less na cl cause prevent to the cell to become swell up.. And no opening of maxi anion channel.. Thus no cl efflux via this channel..that little cl thet enters remain in the cell making the cell hyperpolarize and no activation of penexxin channel.. Also that low cl amount activate COX that convert AA into PGE2 that pge2 act on ep4 receptor via gs pathway on affernet arteriole smooth muscle leads to vasodilation and aslo they cause mesengial cell relaxation..and those pge2 act via ep4 gs pathway to activate the JG cell to release renin.. And that low cl that retained activate the NoS in mdc and release no by mdc to cause affernet arteriole vasodilation.. ❤

Ohk let me explain.. When the map goes high.. From normal upto 200 mmhg.. The renal arterial pressure goes high.. Thus rbf increase.. And thus gfr increase b.. Thus increase in filtration and thus increasing in nacl I'm the short segment of thick asending limb of loop of henle lined by macula densa.. Then the na cl move into the macula densa cell(mcd)via nkcc2.. And increase in na and cl cause Osmosis and increase in water indise the cell.. Thus activate the volume sensitive anion channel Or maxi anion channel.. Thus cl efllux via those anionn channel and the cell got depolarize and that depolarization activate Penexxin channel.. Thus ato moves out at the basolateral side via penexxin and also maxi anion channel.. Then at the ECM present of ecto atpase that convert atp to adp to adenosine finally.. Then those adenosine act on a1r via gi pathway inhibit jg cell to release renin, adenosine act on affernet arteriole by a2b receptor via Gq pathway and also act a1 receptor to conduct gi pathway to reduce the camp and pka to prevent the pka mediated inactivation of MLCK.. Thus promote affernet arteriole vasoconstriction.. And that adp act on p2y receptor via Gq and adenosine act on a2b via Gq contraction of extra and intraglomerular cell to compress the vessel and reduce the lumen size thus reducing capillary surface area thus reducing the GFR.. That reduction of gfr when bp goes high prevent excess electrolyte and water exceetion and prevent electrolyte imbalance and also prevent Vessel injury in renal circulation 😎also high amount of cl inside the MCD ultimately move out so no cl inside..this no activation of NOS (3) thus no NO release by MCD cell and prevent affernet arteriole vasodilation via NO❤

Mam Thankyou so much, it was much needed right now✨

Gem❤

Mam how do you made this videos… please guide me

Just because of this single video, I subscribed this channel

Child can't have o blood group

What is the relationship between velocity and the pressure along air ways ? 😢

Blood group B

Ma if i didn't get you wrong I think it's blood group B

Never can be O blood group

Amazing video ❤

Wow ma'am . It cleared my doubt. Can you explain what will happen to JVP in cardiac tamponade ? And why kussmaul sign would not be seen in cardiac tamponade?

I always struggled understding this thank u for clearing it ma'am❤

Mam if i m not wrong does the Tension of heart more in dilated heart is on the basis of Frank's starling law

Mam is it eccentric hypertrophy in athletes??

Awesome mam Thank you so much

Why addition of protein do not contribute to concentration gradient change?

Very well explaining of jaundice in short interval of time. ❤

Bakwaas ghatiya vdo

thank u mam

Maam , is autonomic neural system purely motor And second do cranial and spinal nerves both have autonomic neurons

Wowwwww 🎉🎉😊😊 Crisp and clearr