Thankyou ma'am Jaise aap concept explain krte ho toh topics samaj me aata hai 🙏🏻🙏🏻🙌🏻🙌🏻

Thankyou❤

So wonderfully explained.. thankyou so much❤❤

is there any particular sequence in which we should watch these videos

Mam there is more ventilation at Apex right

Maam when are you going to make video on spinocerebellar tracts ?

Maam you didnt make video on spinocerebellar tract?

Thank you ,finally understood this topic .

Superb

Lifesaver

Mam your videos are one of the gem that I found in youtube🥹 continue upload more videos related to physiology. Thank you so much mam for explaining the concept in a easy manner. I am very grateful to you. Hats off great work mam🎉. I have no words to explain the happiness after watching your videos, because you explained everything in a easy and understandable manner mam. I know the motivation for our work is essential to achieve more in our pathway. I will support you mam😊

Mam fromwhere can we get ur notes🙏

Thanks

Is this an adequate summary, Professor? - Filtrate in Bowman’s = isotonic to blood. - In ALOH, solute sent out, increasing interstitum osm. - Therefore, H2O moves out of DLOH, decreasing interstitum osm, increasing filtrate osm - As a result, even more solute is sent out of ALOH = positive feedback loop - Vasa recta, w low osm, encounters ALOH, taking solute out of ALOH = increasing blood osm. - Then, vasa recta, now w high osm, encounters DLOH, taking water out = decreasing blood osm.

Nicely explain ..

Great explanation❤

Compliance is not about expansion, it's about deformation (STRAIN)produced per certain amount of STRESS. In case of lung physiology the deformation (STRAIN) is measured in change in volume/original volume. STRESS is that which causes that STRAIN (change in volume) and is measure of Trans pulmonary pressure [intra alveolar pressure(IAP)-intra pleural pressure(IPP) ] The actual formula for compliance is strain/stress. Strain= change in volume/original volume Stress= trans pulmonary pressure Now into your question Even after expiration lungs still have functional residual capacity of 2500ml air inside all alveoli. So even before inspiration the volume of air in lungs is 2500 ml which is V1 Now after normal inspiration (tidal volume) of 500ml the total volume of air now in lungs is 3000ml = V2 Change in volume= V2 - V1 That equals to (3000 - 2500)=+ 500ml That positive sign indicates the stress is tensile and is stretching in nature. Now come to strain STRAIN = change in volume/original volume(V1= 2500ml) Calculate it STRAIN = 500/2500 = 1/5 Now STRESS= trans pulmonary pressure at end of inspiration { Tpm} Tpm = IAP - IPP At end of inspiration IAP = 760mmhg IPP = 754 mm hg So Tpm = 760 - 754 Tpm = 6mm hg Now come to compliance C = strain/ stress C = 1/5×6= 1/30 [ml/mmhg] The inference is for a stress(pressure) of 30 MMHG the strain (deformation or volume change) is 1ML. So for inspiration the lung compliance is 1ml per 30 mm hg Now let's calculate for Expiration Before expiration that is at the end of inspiration the volume of air in lungs is 3000ml Now V1= 3000ML And now stress is elastic recoil of alveoli which increase pressure and produce change (deformation) After normal expiration (T.V) of 500 ml the final volume V2 is 2500 ML So change in volume is - (V2 - V1) The negative sign indicates the elastic recoil of alveoli is of compressive stress So change in volume is - (2500 - 3000) U will get 500ML Now for strain = change in volume/ original volume STRAIN= 500/3000 = 1/6 (no units) Now for stress (Tpm) Tpm at end of expiration is IAP- IPP IAP at end of expiration is 760 mm hg IPP at end of expiration is 758 mm hg Tpm = IAP- IPP Tpm = 760 - 758 = 2mm HG Now for compliance C C = strain/stress = 1/6×2 = 1/12 [ml/mmhg] The inference is for a stress of 12 mm hg the strain is 1ml So during inspiration for producing a change in volume of 1ml u need 30 MMHG But in case of expiration for producing a change of 1ml u just need 12 MMHG. Final take = for a very little stress if we produce more change it has more compliance Compliance of lungs is higher in expiration than in inspiration bcoz in expiration a less stress(pressure) produces more change in volume That's it

Love you aunty you will forever be famous

Nice one 😭❤❤

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I have watched so many videos about countercurrent mechanism,but i couldn't understand anything,but this video is marvellous 😮❤oh my god i understand every concept about this mechanism easily

B is more excitable because it chronaxie duration is less than that of A

Thank you mam ❤️

Found your channel in last few days ....just amazed by the way you explain 🤍

Fantastic video madam🎉🎉

Very nice

Nice explanation..

Fantastic class

mam your video is amazing but plss rub the pen mark you made on the board, its difficult to understand sometimes. thank you

Why do we want our muscle to be sensitive during contraction in alpha gamma coactivation !?

This was a very nice explanation

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Thankyou ma'am u made it clear❤️🎉😊

Thanks you mam for such good explanation.

Thankyou so much mam! U explained it in a very simplified manner

Thanku so much ma'am

Amazing .. no word for Ur dedication towards Ur Vedio 😊

Just wow ...

Ma'am your channel is criminally underrated!!! You're truly a savior, thank you for uploading such simple videos on complex topics!❤

Literally u r a great teacher🙏

Mam pt is having white coat hypertension, wen checked in home bp is normal...here white coat htn is conditioned or unconditioned response??? Pls explain mam thank u