Hypothesis based in physiology: Although hepatorenal syndrome causes prerenal azotemia and we should expect elevated BUN levels, the liver (which is dysfunctional) is unable to build up urea (throught the urea cycle), thus there is less BUN to reabsorb in the kidney = increased reabsorption of scant urea mimics the appearance of normal values. Saludos desde Ecuador!
@lazymedico7485 ай бұрын
whenever i stuck in a topic ....i jump here to clear my concept....salute to you❤