An overview of the diagnostic approach to the acute onset of shortness of breath.
Пікірлер: 68
@StrongMed Жыл бұрын
A word about the diagnostic framework - specifically the category of "hypoxemia". This is an oversimplification of the pathophysiology, since most pathologies listed in this category result in dyspnea via multiple mechanisms (e.g. reduced lung compliance in heart failure and ILD), of which hypoxemia may not be the most important.
@heatherfernanda1094 жыл бұрын
Finding your channel for my Cardiology and Pulmonology OSCE prep is my PA School Miracle! Thank you so very much!!!
@LocTran-pi7uo4 жыл бұрын
I just want to say thank you for all your works
@drfrost3686 жыл бұрын
Thanks for your great informative videos
@aci.5 жыл бұрын
Thank you Dr. Strong for making these videos!
@iszlaimatyasjeno13012 жыл бұрын
Thanks for this fast, dense, helpful explanation
@iliveonthemoonful4 жыл бұрын
Excellent video with simple explanations! Thank you!
@user-gg1vm2cj1y4 жыл бұрын
I have discussions tomorrow and this will help me more more than my teacher does after I learned , you make it together all , 🥰✋🏻 thnz best teacher 👨🏫 keep it
@jeswinjohnvarghese80867 ай бұрын
damn one of the best videos ive come across thank you sir
@rebbekandahuma2173 Жыл бұрын
Thank you so much. Please make videos on approach to young stroke, myaesthenia gravis, guillaine barre syndrome and myelopathy
@khan35406 жыл бұрын
waooo thnx for such precious videos...looking fwd to abdominal pain approch
@masoodpaki6 жыл бұрын
Great and clear explanation, keep it up.
@HafizahHoshni4 жыл бұрын
Awesomely informative and perfectly explained! Thank you so much! 😊😊 15/9/2019
@suneelsharma17634 жыл бұрын
great videos. plss keep posting
@n4ptune6473 жыл бұрын
thanks i was diagnosed with acute dyspnea yesterday
@mehwishkanwal16212 жыл бұрын
Hi doctor strong medicine Thanks for converting theoretical knowledge into practical approaches by your videos.one request plz Can there b a video showing clinical approach to investigate pancytopenia with underlying cause
@jahangirshah24436 жыл бұрын
Is there any of your traditional videos coming soon? I'm eagerly waiting for them ...
@MuhammadAli-ml9bt6 жыл бұрын
thanks alot sir i wish you were my teacher
@sunving4 жыл бұрын
thank you dr Strong.
@vynguyenthiphuong93245 жыл бұрын
Firstly, thank you for your video. It's very informative and helpful. However, I have a question, I really curious about how you decide to categorise the causes of acute dyspnea by the pathophysiology. I know that you had explained a little bit about it in the video but i still find it unclearly abt the reason why.
@StrongMed4 жыл бұрын
I'm so sorry - just seeing your comment now! I largely chose to categorize the etiologies of acute dyspnea by pathophysiology to provide some variety since the etiologies of chronic dyspnea in the corresponding video are categorized by organ system.
@davidbills7217Ай бұрын
I have dyspnea and every doctor I see just throughs antidepressants at me like that is my problem. My breathing is really labored when I eat and its soooo annoying. I've had this problem for about 4 years now and no doctor has gave me any test like ecg, xray or breathing test. My normal spo2 is 94% and sometimes 97-98 when I take iron pills. My BP average is 110/73 but sometimes my resting PR laying down is 85-95bpm. I will bet this is how I will die when its my time. Not being able to breath is not the best wat to go out...
@pachamuthu70113 жыл бұрын
That's awesome ....tq so much sir
@moradzayed6 жыл бұрын
very great lecture thanks alot
@jamieong6004 жыл бұрын
Thank you so much for this!!!
@hh-zq9io4 жыл бұрын
Million thx...can u please make a video about approach to dysphagea
@rohithkumar34805 ай бұрын
Thank you sir for the informative video.How does one differentiate alveolar vs interstitial opacities on a chest x ray?
@StrongMed5 ай бұрын
The whole linked video focuses on this question, but is summarized near the end here: kzbin.info/www/bejne/o3-vlWd6gNl6qZY
@rohithkumar34805 ай бұрын
@@StrongMed thank you sir for the immediate reply .
@ariaran18716 жыл бұрын
Course crepitations only in bronchiectasis and resolving pneumonia....while fine crepitations are found in acute pulmonary edema and ILD..
@syednajmulhassanshah61866 жыл бұрын
Great Work Sir👍
@littlemiss.s72984 жыл бұрын
What is the possible cause of chronic dyspnea at rest without any other associated symptoms in a pt who is hypertensive (well controlled) and all his cardiac a pulmonary and blood tests are normal . And he is not stressed out
@hc_shogun Жыл бұрын
Thanks!
@CreativeMotionDesignH6 жыл бұрын
Thank you
@walidshater31264 жыл бұрын
Thank you so much
@arslanali14326 жыл бұрын
Kindly make videos on approach to other symptoms as well ct head,chest and abdomen
@mohammadhaghighat743 жыл бұрын
Hi doctor Thanks for your amazing lectures. I was wondering whether carbon monoxide poisoning could also be on the differentials list for acute dyspnea?
@felipepalma78182 жыл бұрын
Carbon monoxide poisoning does not stimulate the respiratory center because the mechanism involves neither hypoxemia, hypercapnia nor acidosis. That is why it is called a silent killer.
@Sherirose15 жыл бұрын
Thank you.
@NickUncommon3 жыл бұрын
Would a diaphragma paralysis also be a cause? Is that then neuromuscular?
@phatproo1342Ай бұрын
Do you have any references? pls
@Al36awi5116 жыл бұрын
beautiful
@user-ii9hm7jh6w4 жыл бұрын
any one can explain why some people dislike these videos, especially this one?
@elizabethodidi74993 жыл бұрын
Except for a deadhead
@Anaben114 жыл бұрын
Very instructional video again, thank you so much! Why is "upper airway obstruction" listed in Miscellaneous? Doesn't it lead to Hypoxemia as well and could therefore be listed there? Thanks so much!
@StrongMed4 жыл бұрын
Upper airway obstruction will first lead to dyspnea by causing increased airway resistance, which mechanically loads the respiratory system before it causes hypoxemia. A patient with an upper obstruction can eventually develop related hypoxemia, but it is a late consequence that implies either profound obstruction, exhaustion of the muscles of respiration, or both. The same phenomenon can be seen in asthma in which an asthma exacerbation accompanied by hypoxemia is extremely concerning for an imminent respiratory arrest.
@ganeshnayak48124 жыл бұрын
Tq verymuch sir ♥️
@edris.alkozi6 жыл бұрын
Superb
@crit-ic6 жыл бұрын
Great!
@youssefkhial67914 жыл бұрын
Hi Dr. Eric .. Is it unusal for asthma exacerbations to cause isolated hypoxemia ?
@StrongMed4 жыл бұрын
Yes. The presence of hypoxemia in an asthma exacerbation suggests either a very severe (i.e. life-threatening) exacerbation, or the presence of a concurrent problem such as pneumonia or mucus plugging.
@Som3D4 жыл бұрын
Intonixia bluroxia fluroxia What the hell I don't get a thing what do they all mean could you please explain we all are not doctors
@candlelight70772 жыл бұрын
Hello sir What will be the immediate care for such patients
@StrongMed2 жыл бұрын
It depends on the cause of the dyspnea.
@jsaf17586 жыл бұрын
It seems that my previous comment contains a minor oversight. "pro-NT BNP" should be NT-proBNP.
@maadyk6 жыл бұрын
Hey, is the Chronic dyspnea video out yet?
@StrongMed6 жыл бұрын
Not yet. In about 2 weeks...
@maadyk6 жыл бұрын
Strong Medicine This video was fantastic BTW!
@maadyk6 жыл бұрын
Strong Medicine Any plans of making a new video regarding resources for the USMLE exam, as an update to the previous one?
@StrongMed6 жыл бұрын
I'm sorry, but no immediate plans for that specific topic. It's hard to keep up to date on USMLE resources.
@jsaf17586 жыл бұрын
A WELL PRESENTED GOOD CONCISE REVIEW. However, it seems to me that a few minor inaccuracies exist. In particular, coarse crackles are considered to be of upper (central) airway origin and result from inspiratory airflow through airway related secretions. They are not regarded to be characteristic of interstitial and alveolar edema that present as late fine inspiratory crackles or rales that is inconsistent with this presentation. Another example is the application of ultrasound imaging of the IVC for increased width or congestion with its noted advantages over naturetic peptides (ie BNP) in reference to heart failure as the possible etiology of acute dyspnea. Evidence does exist for the argument of the application of IVC assessment in terms of comparable accuracy when compared to naturetic peptides (ie BNP, pro-NT-BNP) and the benefit of significantly reduced time to investigate a cardiac origin of dyspnea. However, to my knowledge, this is not implemented in major guidelines for heart failure. Once again, I found that the overall brief review to be quite good.
@dinkokambourov5 жыл бұрын
Well said, fine crackles, rales and crepitations are found in cardiogenic pulmonary edema. Thanks for the video, excellent presentation.