I love to swim, doctor said it is okay. He also said okay to adult tricycle, take it slow and easy, use my oxygen.
@jonnuanez7183Ай бұрын
This is my issue. My time right now is SO compressed, so tight, that my gym and cardio time is almost nil and my lungs are paying the price for it.
@eternalwarrior-yp2qxКүн бұрын
Exercise actually triggers lung conditions. You need to be very careful while exercising. Otherwise, you can make your condition worse and healing will be prolonged.
@edwardschlosser453213 күн бұрын
5 failed spinal fusions. Chronic pain range 7-10 with any kind of movement. 6 heart attacks and a double by-pass last year. PPF since 2023. Retired cardiology researcher. At first I thought this a foolish and wasteful approach. However, not many options so I got my MD who knows my background to prescribe 30 mg of oxy and it takes almost 2 full hours to reach a reasonable efficacy, and than I began a 6 block walk requiring just over one hour. This increased my resting O2 level a full 3% higher than with no exercise, and stabilized them for many hours. I repeated the study without the exercise and data looked like garbage with an average O2 sat 3% less and with much greater standard deviations. It isn't the oxy. It is the exercise. I have found that either a beta blocker or a calcium channel blocker can seriously increase the standard deviation. Losartan does not have this effect. What was my most unusual finding. The destabilization of these common cardiac meds can be corrected with 2-3 mg of alprazolam. I discovered this purely by accident and only put on the continuous pulse oximeter because I feared respiratory reduction and low O2 sats. Boy was I shocked and have no reasonable mechanism of action. It eliminated the wild swings to a beautifully smooth curve over very little variation, had no effect on HR , no drops over 3 or 4% and increased my O2 sats by 3%. The only perfect 10 score I've gotten. Average O2 sat was 97%. When is someone going to study the effects of opiates with and without exercise? When are they going to look at the effects of other cardiac meds on O2 sats? Why is a benzodiazepine acting this way. It is not how I was taught in pharmacology 40 years ago. I thing we need studies into the effects of beta blockers and calcium channel blockers on O2 sat stabilization, and come up with some explanation that makes sense as to why a benzodiazepine is acting this way. This could buy patients more time before oxygen becomes necessary, and losartan has the advantage of anti-fibrotic properties.