You shine Luz, just like your name :) Keep up the amazing research and God bless
@luzmilbethcumbagarcia62773 жыл бұрын
Thank you so much! :) I appreciate it!
@luzmilbethcumbagarcia62773 жыл бұрын
Let me know if you have any questions, post them in the comments! Follow me on social media and subscribe to the iBiology channel! Website: luzcumbagarcia.com/ Twitter: twitter.com/lmilbeth Instagram: instagram.com/scitopia/ LinkedIn: www.linkedin.com/in/luz-milbeth-cumba-garc%C3%ADa/
@mohit_102 жыл бұрын
Thank you mam 🙏
@zu30352 жыл бұрын
Madam can you share your Gmail with us please. It will be great favour
@pinnacleexpress4202 жыл бұрын
Do you really know what the EV profile is capable of doing though? What if the immune response inducing cytokinases missing in infected patients would only usher in inappropriate responses that would make the situation worse? For example, attracting immune cells that release superoxides to destroy bacteria walls. What if the immune system just wants to be extra sure that the wrong approach isn't taken? Is there some proof that the virus is even affecting the EV protein ratios? If it was it seems to me like the best direction to go from there is shutting down whatever it is in the virus that's creating/destroying/changing EV ratios, like how doxycycline renders proteins in bacteria unusable, and then seeing if it passively decreases tumor sizes or growth
@Dr_Sri_Harsha_Guthikonda2 жыл бұрын
hi, thank you for the nice talk. I have a small query, i am under the assumption that IL-10 and IL-13 are in general anti-inflammatory. does that seem contradictory to your hypothesis or do these cytokines have a specific role in the surveillance of GBM?