Diabetes Insipidus and SIADH | Clinical Medicine

Рет қаралды 39,049

Ninja Nerd

Күн бұрын

Пікірлер: 58

@abdiwelihassan5310 9 ай бұрын

I was worried you might be gone from this channel forever. But I kept the faith. Welcome back, man. 🙏

@samra6840 9 ай бұрын

I love the diagnostic algorithm part at the end. It really helps with consolidating the information. 🙌

@camilasalcedo9469 9 ай бұрын

ur carrying my entire MD on ur gd back... my tuition should go to u, my guy

@31dharachhatrara62 9 ай бұрын

Beautifully explained with crystal clear concepts , Thank you so much sir 😊, your videos are very informative and helpful to us ..😊

@Conspii_Donald 8 ай бұрын

Why are Thiazide diuretics used for treating nephrogenic DI although they promote diuresis...?🤔

@NinjaNerdOfficial 7 ай бұрын

Great question - they lead to mild hypovolemia which activates the RAAS which leads to enhanced sodium and water reabsorption in the PCT which leads to less polyuria. Although it seems paradoxical it does seem to be effective in nephrogenic DI

@Conspii_Donald 7 ай бұрын

@@NinjaNerdOfficial Thanks boss...🙏

@SoFarAway450 5 ай бұрын

@NinjaNerdOfficial I had this exact same question too and loved your explanation of it here as well. I have a follow up question to add on to it if that's okay. If the cause of the nephrogenic DI is hypokalemia or hypercalcemia, then I assume you correct the abnormality (and/or underlying cause) and you solve the issue. But in instances of CKD or inheritance, do you keep them on long term thiazide tx? Lifetime?

@mukhtasirahmad1875 5 ай бұрын

Incredible the best way of teaching ❤❤❤ but please come to the whiteboard bcz we love your diagnostic explanation 😊

@valviv 9 ай бұрын

Great video, thanks Ninja Nerd!

@mstur-k9s 7 ай бұрын

Love this guy so much ❤️❤️❤️❤️❤️🤌🏼🤌🏼🤌🏼🤌🏼🤌🏼🤌🏼

@abbygonzalez8518 8 ай бұрын

Thank you so much! you made this topic so easy and understandable!!! THE BEST!!!

@reignlulubell 7 ай бұрын

Thank you for your video. I learned a lot even though, I'm student nurse.

@pietropanza8247 8 ай бұрын

So osmotic demyelination syndrome can occur both in an acute and chronic hyponatremia due to SIADH?

@Dr.Number01 7 ай бұрын

INCREDIBLE TEACHER!

@excuseme1139 8 ай бұрын

could you get renal failure in DI?

@bovanovsky 6 ай бұрын

Sir. You're my hero

@FEATHERMAN11485 9 ай бұрын

I want to shake your hand in person after getting this Medical degree!

@JosephFrancis-i7x 4 ай бұрын

In complications of SIADH: The increase in aquaporin channels increases re-uptake of water from the urine, which leads to dilution of blood, and thereby, hyponatremia. But the increased water retention would lead to decreased aldosterone (to counteract the hypervolemia), which would promote sodium excretion in the urine. Water follows sodium, so more water should go into the urine, but since ADH is increased, it just refuses to let that water go, and continues the pattern. How then, would euvolemia be achieved? @ euvolemic hyponatremia

@aravindnarayan7402 26 күн бұрын

thanks

@spectre-24J 6 ай бұрын

great video as always, thank you very much!

@muhammadhuzaifa6352 2 ай бұрын

Please teach diagnostic approaches on board

@قناةمحبيالشيخعليحسينجبر 7 ай бұрын

Thank you alot for this effort ❤

@narjesalbazi7190 6 ай бұрын

Thank you so much ☺️

@Happy-yf8bc 4 ай бұрын

Can't we tell the difference of Diabetes Insipidus versus Psychogenic Polydipsia by looking at just serum osmolarity and urine osmolarity?

@fatmamohamedasefi1604 6 ай бұрын

Thank you so mutch

@saraabuhusein7278 8 ай бұрын

thank you❤️❤️

@muqtarjamaegal6071 4 ай бұрын

Why sheehan causes effect Why not other hypoperfusions don't cause like blood loss injuries and so

@Nurse-Jenni 2 ай бұрын

SIADH causes peripheral edema and high blood pressure?

@yr8563 15 күн бұрын

It doesnt cause edema because although there is an increase in blood volume natriurisis still happens normally and that prevents the creation of peripheral edema.

@Maryaamn 7 ай бұрын

Thanks ❤

@jordanchung1399 4 ай бұрын

Bless bro 💯

@ralph7781 3 ай бұрын

So how do I cure this???? Please help!! It’s ruining my life

@samiaridhi2668 4 ай бұрын

Plz do make videos on solving uworld

@alifakih3840 7 ай бұрын

Great video

@nijabudeenmohammedn5354 8 ай бұрын

how to cure this disease cdi?

@your_therapist_6 3 ай бұрын

His biceps🔥

@sanjith1179 9 ай бұрын

I st like sir, I'm pratheesh from india( tamilnadu).big fan of you sir.say one hii sir.

@3moory-rr2wx 9 ай бұрын

@priyadharshinis4562 8 ай бұрын

BMP= Basic Metabolic Panel

@amanueltadesse9190 2 ай бұрын

Tnx

@sofiaespinosamorales109 9 ай бұрын

TE AMO

@lytonchilembo 5 ай бұрын

you are the best buddy.🤭🙏

@NinjaNerdOfficial 5 ай бұрын

You are the best

@Tuna-z2b 4 ай бұрын

Salt tablets have no effect in treatment at all, every nephrologist will get a heart attack if you administer them to your patients. Fluid restriction is key to recompensate the normal state of sodium levels in chronic and/or asymptomatic patients if the Na is between 120-135 mmol/l. If acute or symptomatic the i.v. treatment is the way to go but as slowly as possible, max 10 mmol/l per day. Otherwise you risk an irreversible central pontine myelinolysis.