Understanding the Real Causes of Heart Disease With William Davis, MD

[@drmorgannolte]

Thanks for watching! To take our Insulin Resistance Diet Starter Course to learn what to eat and why to lose weight with insulin resistance, go to www.zivli.com/Insulin-Resistance-Diet-Starter-Course.

[@pbziegler]

This is, far and away, the best discussion on this subject I have heard to date.My Kaiser dr, a terrific person, wanted me on statins because at 82 I am at risk of dying in the next ten years. Brilliant. I said I didn't want to take statins and I wanted a CAC scan. She said no reason, just take the statins. I was taking them but that was reason enough for me to start researching. I have stopped the statins, cut out processed carbs, eat a Ketogenic diet, exercise (which I have always done). Lost 18 pounds already. The statins put me in pre diabetic range. Now my fasting glucose is 72. No sugar, no grain, carbs from non starchy veggies, fats (no seed oils) and protein from meat, fish and poultry. Occasional nuts and cheese. And occasional glass of wine. Funny, I did this more out of outrage at the medical system than to be healthy. But that's my way I guess. Thanks for this video. It confirms my experience. Side note: There is no such thing as false vs good science. There is science and there is marketing.

[@tiiiip]

Bravo!

[@stevekelly7488]

I am hearing researchers say that seed oils are the worse than surgar. Listen to Dr Cate Shanahan. She is an Family Practice MD that also has a a bio chemistry degree. She believes that sees oils are the real tobacco scandal of our time.

[@niranjanpaul2176]

Sure pufa omega 6

[@skyblueroan]

The lesser of two evils. Added sugars, fruit juice, seed oils. I avoid them all.

[@skyblueroan]

And, so glad to see that a family doctor is advocating for healthy eating!

[@jamesgordon8867]

20yrs working in healthcare facilities. He is absolutely correct! They don't want to correct underlying issues!

[@tinap1111]

Dr. Davis is really helpful explaining all of this. Thoroughly enjoyed this interview. I didn't know about small LDL! Ordered the NMR test! I'm already feeling a bit better about my outcome.

[@StevieRenee-o4u]

One of my favorite online doctors:)

[@ricardorodriguez-mi2zv]

So the key here is to lower consumption of grains, sugars and wheat???

[@realsteel8113]

And eat red meat. Works for thousands of people I know including me.

[@niranjanpaul2176]

Pufa, gmo,

[@chuckleezodiac24]

yep. avoid/limit grains, sugar, seed oils, bread, pasta, rice, potatoes. also junk food, fast food, restaurant food and ultra processed food.

[@ritap3071]

Thank you so much for putting on such an informative and life saving video. Dr. Davis is very easy to understand and gets right to the point.

[@diannalocke997]

Excellent! Big THANK YOU to Dr Davis!!!!

[@richard01983]

If study results made any sense they wouldn't be news. I read "Wheat Belly" and lost visceral fat by cutting out wheat and added sugar.

[@humbleduke6745]

This discussion had me hooked 7 minutes in!! 🔥🔥🔥

[@TheAnimalBasedCure]

Great conversation, I want to know more about every one of those topics you brought up for the next interview!

[@daniellee8866]

Dr Davis, you seem to think that small dense LDL particles, if increased, are the direct cause of atherosclerosis by entering the coronary artery wall to form plaques. I beg to differ. I think the amount or number of small dense LDL particles is merely an indicator of poor metabolic health caused by the excessive intake of wheat, grains and sugar. This indicator is merely an indicator, but it does not mechanistically form the plaques themselves. It is the poor metabolic health, eg the high blood sugar, high blood insulin, low nitric oxide, etc that cause endothelial damage and inflammation; and plaques are actually the result of healed blood clots that form at the sites of endothelial damage. The cholesterol in the plaques was there to heal the endothelial damage during the clotting process. Statins are useless because they do not treat the cause of the disease. If we have a molecule to somehow reduce small dense LDL particles, it is also useless because small dense LDL count is merely an indicator. We can’t get healthy by treating indicators. Indicators merely indicate. We need to correct the metabolic dysfunction!

[@jamesgordon8867]

When are more going to see & evaluate like your comment? We need more data so maybe Primary Care will listen

[@randyblanton5580]

Read Dr. Malcolm Kendrick's book "The Clot Thickens."

[@hiraijo1582]

Thank you for this comment. Retired MD here. Sadly pharma loves to treat indicators. There is a lot of profit.

[@scienceislove2014]

​​@@hiraijo1582are you an md? Then shame on you...becuz you even lack the understanding of basic established science.. I mean..the amount of misinformation this guy has spewed there is simply not tolerable.. and very misleading ..statins don't just reduce cholesterol..they also reduce the no. Of deaths from cvd.. plus sat fat worsens apob which is an important marker of cardiovascular health.. And what's the thing with "clotting process" and "plaque actually heals".. Do they even know what's clotting?

[@scienceislove2014]

The statement contains several elements that are inaccurate and also includes significant misunderstandings and oversimplifications about the role of small, dense LDL particles in atherosclerosis and the treatment of cardiovascular disease. Misinterpretation: The claim that small, dense LDL particles are "not the direct cause of atherosclerosis" and merely an "indicator" of poor metabolic health is incorrect. While small, dense LDL particles are indeed associated with poor metabolic health (such as insulin resistance and high blood sugar), they are also actively involved in the process of atherosclerosis. These particles are more likely to penetrate the endothelial lining of arteries, become oxidized, and contribute to the formation of plaques. Mechanistic Role: Small, dense LDL particles are not just passive indicators; they play a direct, mechanistic role in the development of plaques by promoting inflammation and lipid accumulation within the arterial wall. Poor metabolic health, characterized by high blood sugar, high insulin levels, low nitric oxide, and other factors, does indeed contribute to endothelial damage and inflammation, which are key drivers of atherosclerosis. However, the idea that small, dense LDL particles are merely markers of this metabolic dysfunction and not part of the pathological process of plaque formation is an oversimplification. Both metabolic dysfunction and the presence of small, dense LDL particles contribute to atherosclerosis. The statement that cholesterol in plaques is there to "heal" endothelial damage and that plaques are just "healed blood clots" is misleading. While cholesterol can be involved in repair processes, its accumulation in arterial plaques is a major factor in the progression of atherosclerosis. Over time, these plaques can lead to artery narrowing, instability, and potentially life-threatening events like heart attacks. The claim that statins are "useless" because they don't treat the root cause of the disease is not supported by scientific evidence. Statins are effective at reducing LDL cholesterol (including small, dense LDL particles) and have been shown to lower the risk of cardiovascular events. They also have anti-inflammatory effects that can help stabilize plaques. While addressing the underlying metabolic dysfunction is important, statins play a critical role in managing cardiovascular risk. The dismissal of treating "indicators" like small, dense LDL particles is not accurate. Effective cardiovascular risk management often requires addressing both the underlying metabolic issues and directly reducing atherogenic particles, such as small, dense LDL. Your statement contains some valid points about the role of metabolic health in atherosclerosis but also includes significant inaccuracies and oversimplifications. Small, dense LDL particles do play a direct role in the development of atherosclerosis, and managing their levels is important for reducing cardiovascular risk. While improving metabolic health is essential, dismissing the role of LDL particles and statins in cardiovascular disease management is not supported by the scientific evidence.

[@leslietroy2869]

We won't make changes as long as we insist on seeing malpractice as "WELL INTENDED". The only good intention from the doctors point of view is how much money is in it. To evade the truth is just as evil as not caring about the truth.

[@jamesgordon8867]

Wheats, grains and sugar: all make my blood sugars go up! Why I avoid! Need fats to lose body fats.

[@irenekim8481]

What does Dr Davis think about sourdough made with Khorasan or other ancient grains?

[@DrJK-wm9ec]

In your next podcast can you address the importance of the health of the glycocalyx and the endothelial lining of your blood vessels. It is damage to these that cause atherosclerosis. And SUGAR damages the glycocalyx (no surprise!).

[@jamesgordon8867]

Glad you mentioned glycocalx

[@johnsavage4786]

Read The Clot Thickens by Dr Malcolm Kendrick this is another fantastic read

[@MrUncleBob]

hmmmm, very very interesting 🙂

[@robertkelly2823]

How does blood donation affect cvd

[@canalroadadventures5079]

I have apo4/3 and the only way I have found to reduce the sdLDL is to take a statin, unfortunately. I eat few carbs and no wheat/grains and my triglycerides and insulin are low .

[@johnalice2657]

This gets more confusing because Cleveland heart labs has separate tests for small, dense ldl and oxidized ldl, in addition to small ldl. Clear as mud.

[@azdhan]

Really interesting. Nicholas G Norwitz, together with Dave Feldman, and hughly reputable and respected Lipidologist, William Cromwell are carefully researching the issue of what increases your risk of CVD the most, insulin resistance leading to cardiometabolic vulnerability or high LDL/ApoB in the absence of insulin resistance. It will be interesting to see what they discover and its application to the general population. Right now the research is based on a hypothesis that in certain people who meet the eligibility criteria of Lean Mass Hyper Responder and are following a ketogenic diet, high LDL may only be a ninimal artifact abd their insulin sensitivity and avoidance of T2D may put them at minimal risk. Earlier research they conducted looking at heart scans of participants found zero arterial calcium scores and no evidence of even softer plaque. This makes me wonder if the Lean Mass Hyper Responder Phentoype may be what Dr Davis has refereed to in another videos as the “Perfect Carnivore” And a super high carb diet or carb loading could have deteimental hard health outcomes on this phenotype

[@scienceislove2014]

You need to watch his interview with Dr. Gil on the Nutrition Made Simple channel.

[@scienceislove2014]

I dunno how this guy even is a doctor. Sat fat worsens apob and a doc should know that but still he's after all sorts of grains (including whole grains ,which is wild!) while saying bacon is not the issue.

[@scienceislove2014]

Dr. William Davis's claim that ApoB wouldn't be affected by the amount of bacon (or other high-fat foods) you eat is questionable and doesn't align with the broader understanding of how diet influences lipid levels, including ApoB. Understanding ApoB and Diet: What is ApoB? ApoB (Apolipoprotein B) is a protein found on the surface of all atherogenic lipoproteins, including LDL, VLDL, IDL, and Lp(a). The level of ApoB in the blood is a direct measure of the number of these particles, which are associated with an increased risk of cardiovascular disease (CVD). Dietary Impact on ApoB: Saturated Fats and LDL-C: Diets high in saturated fats (like those found in bacon) can increase LDL cholesterol (LDL-C) levels in many people. Since LDL particles carry ApoB, an increase in LDL-C generally corresponds with an increase in the number of LDL particles and, therefore, ApoB levels. Triglycerides and VLDL: High intake of saturated fats and refined carbohydrates can also increase VLDL (very-low-density lipoprotein) levels, which are rich in triglycerides. VLDL particles also contain ApoB, so an increase in VLDL could lead to higher ApoB levels. Individual Variation: There is some individual variation in response to dietary fat. Some people might not experience significant changes in their ApoB levels after consuming foods high in saturated fats, but this is not the general case for the majority of the population. Bacon and Cardiovascular Risk: Processed Meats: Bacon is a processed meat that is not only high in saturated fat but also contains high levels of sodium and preservatives like nitrates, which have been associated with increased cardiovascular risk. Regular consumption of processed meats has been linked to a higher risk of CVD, possibly through mechanisms beyond just ApoB levels. The suggestion that the amount of bacon you eat wouldn't affect ApoB levels runs counter to the broader scientific consensus, which acknowledges that diet, particularly saturated fat intake, can influence ApoB levels and overall cardiovascular risk. Dr. Davis’s claim that bacon consumption wouldn't affect ApoB levels is not well-supported by current scientific evidence. Saturated fats, such as those found in bacon, can increase LDL cholesterol and ApoB levels in many people, contributing to cardiovascular risk. It's important to consider overall dietary patterns and prioritize heart-healthy foods to manage ApoB and reduce CVD risk.

[@scienceislove2014]

Dr. William Davis is a trained cardiologist, but his credibility in the broader medical and scientific community is mixed due to his controversial and often unscientific claims. While some individuals may benefit from his dietary advice, especially if they have specific intolerances or conditions, his blanket demonization of wheat and grains is not supported by the majority of scientific research. Criticism from the Scientific Community: Selective Use of Data: Many experts have criticized Dr. Davis for making broad and sometimes unsubstantiated claims based on selective data. For example, while there is evidence that refined carbohydrates, including products made from refined wheat, can contribute to metabolic issues, Dr. Davis's stance against all forms of wheat, including whole grains, is not supported by the majority of nutritional research. Lack of Peer-Reviewed Evidence: The ideas presented in "Wheat Belly" and some of his other works are not widely supported by peer-reviewed studies. Mainstream nutritional science generally supports the consumption of whole grains as part of a balanced diet, which is contrary to Davis's recommendations. Oversimplification: Critics argue that Dr. Davis tends to oversimplify complex issues, like obesity and heart disease, attributing them primarily to wheat consumption. These conditions are multifactorial and cannot be pinned down to a single dietary component for most people. Supporters and Popularity: Despite the criticism, Dr. Davis has a large following, and many people report positive health outcomes after following his advice, particularly those who might have had undiagnosed gluten sensitivity or other wheat-related issues. However, anecdotal evidence like this is not a substitute for scientific validation.

[@brendanokeeffe3509]

On requesting my GP I got a CAC Score of 4000 ❤ .I was told by GP that, having had a quadruple bypass 30 years ago ,such a result did not have any predictive value Could not get an explanation ?? Can you explain? Many Thanks Brendan O Keeffe

[@jamesgordon8867]

Insulin levels

[@shawnfallahi5616]

what is your argument for someone to NOT take a statin, if they have suffered a heart attack (stents placed)?

[@Davidkxf]

My trigs are very high and I'm 95% carnivore for over 8 years now.

[@BrendaKing-zs9tb]

Thats weird

[@chuckleezodiac24]

this used to happen to Dave Feldman. he calls it the "Triglyceride Carryover Effect." he has videos and blogs related to it. i think it depended on how many hours he had fasted before the blood tests. his TGLs dropped to normal 1 or 2 hrs after he would wake up. kzbin.info/www/bejne/onvHeXl5bq1qj7c

[@mpoharper]

My triglycerides went up on statins. Keto without statins they go down.

[@UnknownUser-sc6jx]

More fat less protein and try eating fish like salmon and sardines for the omega 3s

[@ClareBoyd-f8c]

Wilson Patricia Martinez Margaret Williams Michael

[@scienceislove2014]

Dr. William Davis is a trained cardiologist, but his credibility in the broader medical and scientific community is mixed due to his controversial and often unscientific claims. While some individuals may benefit from his dietary advice, especially if they have specific intolerances or conditions, his blanket demonization of wheat and grains is not supported by the majority of scientific research. Criticism from the Scientific Community: Selective Use of Data: Many experts have criticized Dr. Davis for making broad and sometimes unsubstantiated claims based on selective data. For example, while there is evidence that refined carbohydrates, including products made from refined wheat, can contribute to metabolic issues, Dr. Davis's stance against all forms of wheat, including whole grains, is not supported by the majority of nutritional research. Lack of Peer-Reviewed Evidence: The ideas presented in "Wheat Belly" and some of his other works are not widely supported by peer-reviewed studies. Mainstream nutritional science generally supports the consumption of whole grains as part of a balanced diet, which is contrary to Davis's recommendations. Oversimplification: Critics argue that Dr. Davis tends to oversimplify complex issues, like obesity and heart disease, attributing them primarily to wheat consumption. These conditions are multifactorial and cannot be pinned down to a single dietary component for most people. Supporters and Popularity: Despite the criticism, Dr. Davis has a large following, and many people report positive health outcomes after following his advice, particularly those who might have had undiagnosed gluten sensitivity or other wheat-related issues. However, anecdotal evidence like this is not a substitute for scientific validation.

[@jamesmcgarity614]

Now you know why I didn't get anymore COVID shots

[@fa4615]

Sorry you are not letting your guest to say what he wants you comments to much

[@allencrider]

Sure, William Davis has the truth. EVerybody else is a liar.

[@donaldobrien9171]

There are many other docs that agree with Davis. Ken Berry, Ford Brewer, and many others, some of whom are in other comments

[@sabeelhoneycom]

Always love to listen dr. Davis. One thing i noticed he talks like trump. No pun intended. But similarities end right there. He is a great doctor and we like him as he is.

[@BrendaKing-zs9tb]

Knpw what i cant get my cardiologist to do an NMR for me or a CAC

[@divadyrdnal]

I had to pay for my NMR myself using “Own Your Labs” web site…BTW I found out I have lots of small LDL-P, I’m already not eating sugar/wheat/grains for many years

[@UnknownUser-sc6jx]

Lie to them tell them a close relatives had a heart attack or heart disease early some times you have to bend the truth a Little, that's how I got my CAC test and advanced lipid profile

[@debdroessler5199]

Just pay for it yourself mine was only $110. Otherwise get a new Dr!!!!

[@BrendaKing-zs9tb]

Ok how to get it without a dr​@@debdroessler5199