Easy way to remember: Life of PAI(plasminogen Activator Inhibitor) begins in the morning! Cause of increased thrombotic strokes/MI in the early morning hours
@asamvav2 жыл бұрын
I liked Dr. Baloor's way of questioning and calmness in explaining. Very good for an examinee. Good presentation from the student's end.
@sakshikumari7349 Жыл бұрын
What is the dofference in progression of hemorrhagic and thrombotic stroke?
@Little-v2x10 ай бұрын
@@sakshikumari7349The difference is time of occurance..progession almost looks similar.. Also can be differentiated on symptoms headache, vomiting in hemorrhagic stroke is classical
@tawfiqahmad80826 ай бұрын
Before watching the video completely, i want to take a minute to appreciate Ms. Rekha. You are you going to be a great doctor. You almost answered every question. I am soo happy for you.
@parthkulkarni83093 жыл бұрын
Mam almost answered all questions…. 😱😱😱
@krisshhnaa50742 жыл бұрын
😱😱😱
@dryash8662 жыл бұрын
That's bcoz she already heard sir's class
@tejasolanki59992 жыл бұрын
@@dryash866 which class??
@medinindia885 Жыл бұрын
Namaste sadguru 😁
@natarajumididoddi598511 ай бұрын
It is a very good clinical case presentation and discussion. I am a family practitioner, (practising)76y.I appreciate the student' presentation .I learnt a lot about hemiplegia, how to diagnose ischemic haemorrhagic and embolic strokes. I wish I were your student. Thanks
@moinalijanvekar9391 Жыл бұрын
Salute to the girl she is very knowledgeable
@___s___807110 ай бұрын
1:07:43 CADASIL is Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy
@manojk6531 Жыл бұрын
Early morning presentation of thrombotic stroke is due to Cortisol rise in body which cause vasoconstriction leading to Stroke is what a professor in my college had said
@drsiddharthsoniphysician2 жыл бұрын
Mesmerising presentation and very insightful and indepth discussion by Bolloor sir who is a gifted teacher. Voice of presenter is very clear and soothing type. @
@dr.hitendrakumar9338 Жыл бұрын
Yes boss 👌
@tarunjha32574 ай бұрын
Hands down the best case presentation i watched ever in my life
@ijaziqbal15392 жыл бұрын
great teacher ..... teaching from heart....... best source for clinical exam prepration ....... true name of teacher
@___s___807110 ай бұрын
1:12:11 stroke mimics SOL Migraine Meningitis Hypo/Hyperglycemia Todd's paralysis
@anasahmad67493 ай бұрын
24-7 hr in Todd's
@meenakshi63443 жыл бұрын
Very useful. Plus Ms Rekha did brilliantly.
@anandtiwari52 Жыл бұрын
Admin, succeeded in maintaining the communication.......good quality audio and video this time, weldone Sir.
@nsas955 Жыл бұрын
Really an amazing and fruitful presentation . I admire the way of presenting the case as well as the questions and discussions. God bless you both.
@kapilchandrothiya33166 күн бұрын
Wow What a Case Presentation 🎉Dr Boolor Sir is Amazing🤩 Must watch !!
@satyamp6202 Жыл бұрын
Thank you so much entire team, great efforts 🙏🙏🙏🙏, thank you so much sir for your valuable time....
@anirbanpaulrollno60679 күн бұрын
Thanks sir for explaining the concept in a very nice way ❤
@breezeofease63732 жыл бұрын
sir pls bring the case presentation over parkinsonism with archit baloor sir ... the way he explain helped me a lot while my case presentation 💓💓💓
@anasahmad67493 ай бұрын
1:02:08 htn Ischemic > hemorrhagic but both possible Lacunar infarcts very small vessels .3-1.5 cm size
@anasahmad67493 ай бұрын
Pure motor Or pure sensory Or hemiballismus Lacunar infarcts Post limb of IC >> genu, thalamus
@narasimha78703 жыл бұрын
Why UMN lesions have distal muscle weakness first any logic? Because if UMN supplying proximal muscle involved ,then patient will have only proximal muscle weakness…..
@nadeem3rashid3 жыл бұрын
Wow ! Very Well presneted for a UG 👌🏼
@itsgabriel26913 жыл бұрын
Doing a great job sir 🙏🙏🙏 keep posting S Help a lot of solving doubts Nice discussion sir ma'am give aprrox all the answer
@anasahmad67493 ай бұрын
1:26:47 ischemic stroke don't reduce bp achanak se as penumbra ko bachata hai Redce slowly While hemorrhagic stroke eg if bleed..reduce
@tube4429 ай бұрын
She doesn't look like undergraduate
@gajananrepal38472 жыл бұрын
Thank you so much Archit sir .....very comprehensive
@pramodgowdar97603 жыл бұрын
Ms Rekha answered everything but lill anatomical basics and surface anatomy she needs to know ... Thats it
@anandtiwari52 Жыл бұрын
Discussion has been thorough and so very valuable.
@BharathKumar-we3ng2 жыл бұрын
Omg is it undergraduate student who presented
@rizvyrahman38373 жыл бұрын
Thanks❤... Archit sir is as always great
@arpanuppal00073 жыл бұрын
Such an amazing discussion ❤️
@anasahmad67493 ай бұрын
1:28:40 increased ict pe decreased RR + Certain soecific patterns eg cheyne stomes etc
@pranavlokeshgp833 жыл бұрын
Really good! Gonna prepare for my case based on this 👍🏽
@sayanthvishnusubramanian88933 жыл бұрын
😅
@natarajumididoddi59855 ай бұрын
Sir in my experience I witnessed in icu (I work as a duty doctor now as well) a few traumatic SAH.
@anasahmad67493 ай бұрын
LOC hemorrhagic or large infarct Cortex and RAS (brainstem ) involved in alertness consciousness
@anasahmad67493 ай бұрын
Doubt..Deviated face towards ? 27:50 Umn cl Lmn il Brainstem lmn opposite side of stroke Above cortical on side of stroke
@SB-uc8ph8 ай бұрын
Beautiful🎉❤
@mdmillatreza950111 ай бұрын
Amazing discussion
@akv41av3 жыл бұрын
Nice presentation n discussion 👍
@AshokKumar-bm4bp3 жыл бұрын
Thank you very much archit sir 🙏
@anasahmad67493 ай бұрын
1:15:08 do not forget autonomic fn...asked for lateral medullary synd
@anasahmad67493 ай бұрын
Horners mainly
@anasahmad67493 ай бұрын
Cervical cord inv(rarely)
@anasahmad67493 ай бұрын
Timing Deficit at onset Progression For type of stroke based on history
@anasahmad67493 ай бұрын
1:18:20 relevance of pulse in stroke...irregularly regular pulse in a fib Raised ict decrease?? in pulse Bradycardia cushings reflex Arterial wall palpable and thickened in severe atherosclerosis Peripheral pulses absent feeble in vascular diseases and variuous cardiac diseases... valvular ds AR - WATERHAMMER AS - ANACROTIC pulsus parvus et tardus Pulse mein rhythm volume character and arterial wall ke ilaawa also auscutate CAROTID bruit
@anasahmad67493 ай бұрын
Also for posterior circulation ... vertebral artery(from subclavian 1st part) mei check bruit...straight line from medial end of clavicle and mastoid... surface anat. Of vertebral artery and arising from there it goes up thru transverse foramen of vertebra
@anasahmad67493 ай бұрын
No bruit only rules out ...possible ke complete occlusion ho jayega
@anasahmad67493 ай бұрын
No bruit in complete occlusion....bruit in 30-70 percent occlusion 1:25:23
@anasahmad67493 ай бұрын
1:28:04 watershed infarct- in multiple areas
@sakshikishen3750 Жыл бұрын
Great discussion!
@anasahmad67493 ай бұрын
1:35:57 Spasticity not rigidity bcz only antigravity muscles i.e flexors in upper limb and extensors in lower limb affected
@anasahmad67493 ай бұрын
Rigidity all muscles affected
@anasahmad67493 ай бұрын
Spasticity has clonus+ babinsky sign Spasticity is velocity dependent ...vel increase krne pr spasticity also increases whereasd rigidity increases Spasticity is Clasp knife spasticity pattern while rigidity is lead pipe pattern
@tarunjha32574 ай бұрын
Kuddos to the presenter calm cool and super intelligent
@anasahmad67493 ай бұрын
1:32:23 in fundoscopy subhyaloid space- boat shaped...dursen's syndrome
@narasimha78703 жыл бұрын
Mechanism of fever in venous strokes and why fever doesn’t happen in arterial stroke??
@GauravPawar-vl7jb Жыл бұрын
No deliveryof inflammatory mediators in case of arterial stroke
@rahullinton10 ай бұрын
So much helpful sir❤❤❤❤
@asfiyafakiha10 ай бұрын
Thankyou sir. 😊
@anasahmad67493 ай бұрын
1:16:47 xanthomas etc for metabolic synd,
@anasahmad67493 ай бұрын
Temp high bad prognosis Hyper pyrexia in endocarditis , pontine lesions,meningitis, venous strokes eg due to DVT
@nishithmohapatra7206 Жыл бұрын
Why posterior limb of internal capsule stroke localization
@anasahmad67493 ай бұрын
Aphasia localises to cortex dosorder of language Dysarthria articulation affected brainstem or any cranial nerve eg 7, 9 , 10 , 12
@anasahmad67493 ай бұрын
1:09:56 young female ho toask menstrual history Ocp Apla syndrome history etc
@anasahmad67493 ай бұрын
1:41:49 3 and below power u can't do cerebellar
@anasahmad67493 ай бұрын
Facial nerve(since face dev) involved therefore PPPPP wali sound lips inv.. in sounds mei dysarthria
@anasahmad67493 ай бұрын
Iç lesion not produces aphasia ...therefore 2 lesions
@anasahmad67493 ай бұрын
Word out put females more
@anasahmad67493 ай бұрын
Edh or sdh hematoma may lead to hemiplegia kind of symptoms
@Dr.GAURAV45 Жыл бұрын
Internal capsule involved, so why hemiparesis and not hemiplegia?
@GauravPawar-vl7jb Жыл бұрын
Don't say hemiplegia until zero power
@mahimabhojwani290811 ай бұрын
Is it left side or right side umnl at 1:35:05
@GauravPawar-vl7jb Жыл бұрын
In umn distal weakness start first eg in fingers
@sachinkambar.9816 Жыл бұрын
How sensory components intact if internal capsule involved
@romanchitupadhyaya3738 Жыл бұрын
Can we have a case presentation like this on paraplegia also sir regarding the approach and all 🙏
@THEWHITEARMY Жыл бұрын
we already have many a discussion videos on paraplegia..kindly check
@anasahmad67493 ай бұрын
Raised ict in hemorrhagic, or large thrombotic
@ruhinaresh74102 жыл бұрын
Very useful 👍
@anasahmad67493 ай бұрын
Umn lesion first has distal motor weakness
@anasahmad67493 ай бұрын
Only lower part of face affected therefore UMN type
@lavanyakumhar80212 жыл бұрын
How is this hemiplegia but not hemiparesis
@sakshikumari7349 Жыл бұрын
What is the difference in progression of thrombotic & hemorhagic stroke? Can it be differentiated clinically?
@Little-v2x10 ай бұрын
Both have almost same progression.. But vary in time of occurance.. Symptom wise hemorrhagic will be have raised ict with Heachache, vomiting
@mr.x13633 жыл бұрын
V nice 👍
@Drstrange122332 жыл бұрын
Sir provisional diagnosis i think it is left sided umn facial palsy as on examination we have loss of nasolabial folds on right side so contralateral will be left facial palsy
@AravindRavi Жыл бұрын
i also made the same confusion during my models. if the patient has loss of nasolabial fold on the right side and deviation of angle of mouth to the left. then we call it right sided facial palsy only but the lesion is on the left side (if its an UMN) . its quite tricky but we have to be careful while answering
@anasahmad67493 ай бұрын
1:44:44 treatment
@mohitzambare Жыл бұрын
Can someone explain the planter response why is it like that?
@sanjujohn63833 жыл бұрын
Very good
@ayushmanmukherjee9297 Жыл бұрын
Here, the history of deviation of angle of mouth as taken, will be right sided and not left sided.... M i right?
@medicalknowledge79502 жыл бұрын
Sir,can u make subtitles available for this vedio ? I have some hearing problem
@DrChainSingh2 жыл бұрын
Thank you sir
@wmaperera2 жыл бұрын
Well done sir...thanks u..
@ravibhoria55639 күн бұрын
Who's the girl presenting the case??
@anasahmad67493 ай бұрын
Dense hemiplegia localises to IC
@adarshpal49073 жыл бұрын
Kadak,🤗🤗
@krisshhnaa50742 жыл бұрын
Don’t be like layman man…
@shashankgowda22082 жыл бұрын
Using India language in no way makes u layman .. knowledge is imp
@shashwatchaurasia9030 Жыл бұрын
Hello medicos, Does anyone have notes regarding this video discussion?
LMN FACIAL PALSY WILL GIVE YOU IPSILATERAL PALSY??
@kishanvishwamitra7727 Жыл бұрын
Chassignac tubercle
@drvijayfromkem69343 жыл бұрын
Thank you to all
@ViratKohli-yi1xr Жыл бұрын
Do we have to know everything like here ?
@anasahmad67493 ай бұрын
Rf here Post menopausal Female Old Htn
@prantikdas99742 жыл бұрын
Woww 🔥
@k.charitachowdary1130 Жыл бұрын
1:26 speech
@kalyanirathod86832 жыл бұрын
👏👏
@dr.vigneshwar57472 жыл бұрын
👌
@docchethanr Жыл бұрын
1:07:38
@kirankumarch995411 ай бұрын
I want pdf of this video
@anasahmad67493 ай бұрын
1:34:54 ??
@anasahmad67493 ай бұрын
Tia r/o ke liye transient loss of vison(pertaining to anterior circulation carotid srtery-amaurosis fugax,..opthalmoc art inv.>>> monoparesis, sensory loss aphasia Or posterior circulation vertebrobasal...weakness in iol(cranial nerves) , vertigo (ataxia) hiccoughs If More than 2 tia ..rx. put on anti platelets Tia indicstes ischemic stroke....can be thrombotic or embolic stroke Tia rules out hemorrhagic stroke Now embolic recurrent short lasting deficit different features each time Thrombotic stroke longer duration in bw...vessel is narrowed ..same vessel damaged..same features each time Tia - 7 times increased risk of getting stroke 60-70% will get a stroke Majority will get. A stroke in first week...rest in first 3 months Abcd2 score
@anasahmad67493 ай бұрын
No history of seizures r/o cortical inv
@anasahmad67493 ай бұрын
Meningitis fever vascular inflammation...eg tb (chronic).. may cause stroke??????
@anasahmad67493 ай бұрын
Involuntary inv basal ganglia No urinary and fecal incontinence...seizures...as present in post ictal
@anasahmad67493 ай бұрын
LOS IC c/l Lateral medullary synd Thalamic - burning pain
@anasahmad67493 ай бұрын
LOB -Cerebellar stroke Also commentrd if power normal otherwise power ki wjh se bhi