Do senolytic drugs (Dasatinib+Quercetin) mechanistically work the same way as the body's innate immune system (Killer T-Cells) at targeting and killing only senescence-associated secretome cells (SAS)? How often would you need to do senolytic therapy as a prophylactic against cancer? And if you need tissue repair, how soon after damage would you have to topically inject the growth factors and any other support/signaling agents? Also interesting comment from Dr. Campisi, that the standard C57 black-mouse will have different lifespans, based solely on the water acidity of the lab that ordered it!