The CRITICAL Link Between Mitochondrial Heteroplasmy And Cancer

[@DrCaseyPeavler]

When I found that last figure about the metabolism of mutant mitochondrial metabolism my mind was blown! 🤯🤯 Were you surprised?

[@raykinney9907]

So, if you get into ketosis via very careful keto diet, plus take ketone esters to some degree, thereby cutting back on glucose to approach only having glucose via gluconeogenesis, is that enough to starve off the energy production driving tumor growth? But, then, glutamine remains to be dealt with by cutting back on that energy supply too? Does glutamine fueling produce as much ROS as glucose fueling? If the idea is to reduce fueling by both routes, while also trying to keep excess ROS high (to throw more wrenches into the works), is it looking like all of those conditions are likely to reached, to disadvantage tumor function and growth? What relative levels of ROS could be expected from ketone fueling, compared to glucose or/and glutamine fueling?

[@DrCaseyPeavler]

As always good questions. In general ketogenic diets only deal with the glucose side of things. Although there are many other factors (blue light, nnEMF, stress, poor sleep etc). There are specific interventions for the glutamine side of the equation. We will talk about all of the known interventions during this series.