The single best video I have found on the topic. So many doctors have failed to explain this to me all throughout medical school. Blind memorisation versus proper understanding.
@derbigpr500Ай бұрын
Because they don't understand it, this is stuff understood by people who specialize in it and who encounter patients like these, although we like to believe that we all understand all of physiology, it's just not the case in practice. I wouldn't expect a cardiologist or an orthopedic surgeon to perfectly understand this although they once, long ago, studied it in med school and probably knew it for some test.
@ajh1952 жыл бұрын
Going to my OSCE exam in medicine and this is the best video i have ever seen! Thank you for your time and help clarifying this.
@duclvr2 жыл бұрын
Outstanding review of relevant neuroanatomy and clinical findings. Useful--like all your other videos--in my quest to be a vertigo ninja. Thank you!
@AlexanderRoux2 жыл бұрын
I believe the term is vertigo assassin
@zachbrown25142 жыл бұрын
As a chiropractic student this video has answered so many learning gaps!
@songjkim2 жыл бұрын
Your videos are singlehandedly the best resource for diagnosing and treating acute vestibular syndrome, I thank you!
@sayamunyrattanatay4904 Жыл бұрын
First ENT rotation exam and this solidifies my studying. Thank you.
@FreedomandRights4US2 жыл бұрын
I noticed an interesting phenomena in certain people I talk to that had this seemingly uncontrollable single eye roll when blinking + sighing over some situation. The pursuit of some explanation of that is what led me to the term Nystagmus and this is definitely the best video on that for someone who has never heard of it. Thanks.
@k.c.86582 жыл бұрын
Thank you for these, can never review this stuff enough
@sman5877 Жыл бұрын
Thank you so much for making these videos. I just went to the hospital for vertigo something i never have experienced. if i would have known this information i probably could have avoided my hospital visit. I’m sharing your videos with everyone i know because we are all getting older and will all experience Vertigo eventually. Blessed day Sir.
@PeterJohns Жыл бұрын
I glad you found this video helpful as a patient. However, this video is aimed at educating medical professionals, and not meant to replace assessment by a medical professional.
@steveblomerth Жыл бұрын
I have enjoyed all your videos because as a chiropractor I see patients who are mis-diagnosed in the Emergency Room and/or only given meclazine after having head and neck MRI's and discharged. I have used the Dix Hall Pike and the Epley manuever for many years. But these videos help me know more and do more for my patients and help me learn much more that I need to know. Well done and thank you. I am digging in now to learn more and I appreciate being able to do low tech diagnostic procedures in office as well as have some tool like Epley and Gufoni to help people immediately.....Thank you for your knowledge and thorough presentations.
@PeterJohns Жыл бұрын
I'm so happy to hear that these videos have helped your patients! That's why I make them, so thank you so much!
@steveblomerth Жыл бұрын
@@PeterJohns Thank you for being such a great resource. I had a patient come in recently with a complaint of dizziness from a local Emergency Room and his work up included MRI's of the head and neck, EKG's, and cardio blood work and the report said he had a positive Dix Hallpike. I inquired about how they performed that test and the patient said he just reported that he got dizzy when he turned his head to the left, but no procedure was done to elicit the dizziness or look for nystagmus. An example of high tech being over utilized and low tech ignored. He was actually negative for Dix Hall Pike but positive on the Supine Head Roll towards the right with up-beating nystagmus. I was better prepared to examine and treat by learning from you....At 72 I am still learning....thank you for your help.
@triplevitalitytv42602 жыл бұрын
Instant subscription. It seems your channel will be incredibly useful for my upcoming clinicals -- working with a vestibular specialist and trying to get down some solid foundation beforehand! Thank you!
@dorotanowodworski30402 жыл бұрын
Diagnosed this today! Thank you for your teaching here on KZbin and when you recently came to QCH.
@PeterJohns2 жыл бұрын
Excellent! Glad to hear in person teaching helps as well!
@braoramesh2 жыл бұрын
Thank you very much Sir. Now I understand why the direction of the Nystagmus are opposite in Dix Hallpike test and in HINTS test.
@annaskrzypek10295 ай бұрын
amazing!!! especially the explanatin of why the rapid phase is towards the healthy ear, thank you!!
@skohaya2 жыл бұрын
Thank you for your videos. Please do a video like a flow chart for BPPV. 1. DX posterior 2. Lateral geotropic vs ageo, affected ear, Bppv that had continuous nystagmus on movement, cupulothiasis? Wishing there was just a one video Bppv if this perform Epley if that perform the bbq roll, or then perform the gufoni. One time a PT moved my crystal that got into the lateral. I was stranded for the weekend because she couldn't get me back in. I watch a video and figured it was lateral when on the right side, ageo. Nystagmus pointing upward. Thought it was the right affected ear but saw a video which explained it was the left. Did the gufoni adaption and was healed in 15 min. I just wish there was a step by step DX. To figure out position in canal, affected ear and correct maneuver. There are videos but they are all chopped up. Please make the go BPPV video.
@harrymusson38872 жыл бұрын
UK GP trainee here - great video, thank you!
@TheKCMadrid2 жыл бұрын
Thank you sir, never tired of this topic especially with your concise but comprehensive explanation. Please keep doing this. Also please consider doing a video on pitfalls that must be avoided/careful about and how to follow my patient to make sure I got the correct diagnosis.
@PeterJohns2 жыл бұрын
Thanks, All interesting topics to make videos about. I'll put them on my (already lengthy) list of videos to make.
@pcb-uf5rf Жыл бұрын
Probably the best explanation of this i have received. far superior to my medical school lectures. Thank you so much
@wikunda2 жыл бұрын
I am a physician from Thailand and I find your video very informative and easy to understand. Thank you so much, now I am more confident when treating patient who presents with vertigo. I have one question for you, from your many videos, it seems like your patients cooperate very well with the examination, which I find it really hard to do in Thailand as
@dgen78408 ай бұрын
Thank you , I was lacking a link in the chain which was the brain interpreting more activity on one side vs the other as movement in that direction hence causing the eyes to move , such simple reasoning yet missed by so many professors !!!
@federico4727 Жыл бұрын
Hello Dr. Johns, I am a young neurologist from Italy. I wanted to thank you for the knowledge and the very useful videos you share with everyone. I rewatch your videos often. They have changed the way I look at patients with vestibular problems! I wanted to ask you if you ever happened to perform Head impulse test to evaluate vestibular function in patients who did not have vestibular neuritis. For example in patient with neurinoma of VIII cranial nerve.
@PeterJohns Жыл бұрын
Glad you have found my videos useful. I have never seen a vestibular schwannoma in my practice as an emergency physician, so I can't help you there.
@worldaround6520Ай бұрын
Thanks a lot, sir. There is a mnemonic that states if stress lines (forehead wrinkles) are present, it indicates a serious condition like a stroke. However, if stress lines are absent, it points to Bell's palsy. The reasoning is that when the entire facial nerve is affected, as in Bell's palsy, stress lines cannot form. But because each facial nerve (left and right) receives signals from both sides of the brain, in the case of a stroke, both the facial nerves still get some supply from the brain, allowing for the preservation of stress lines on the forehead. The same concept applies to vestibular neuronitis: in peripheral lesions, there is manifest damage, while in central nervous system lesions, the damage may not be immediately obvious (non-manifest damage). Birds keep their heads stable so they can focus on a target even while their bodies move. In humans, this function is achieved by the vestibulo-ocular reflex, which allows the eyes to adjust in such a way that focus remains on the same target when the head moves. The vestibular system neutralizes the effect of head movement by making the eyes move in the opposite direction to the movement of the head. Keep in mind that eye muscles work by pulling, so the vestibular system detects motion on the opposite side and pulls rather than pushes. When the eyes and ears evolved, if you hear a sound from the left ear, you would want your eyes to move to the left. Therefore, it makes evolutionary sense that the vestibular system pulls rather than pushes. If my right-sided vestibular system is damaged and I move my head towards the right, the left-sided vestibular system will become activated and pull the eyes rapidly towards the left. If both vestibular systems were normal, the eyes would still deviate towards the left. However, because the right-sided counter-pull is absent, the movement of the eyes towards the left will be more rapid, and the eyes will move further left than needed. This question was asked in NEET PG 2024. If the right-sided vestibular system is dysfunctional and I move my head towards the right, the eyes will move rapidly towards the left, moving further left than needed. The brain will then compensate for the loss of right-sided vestibular function, causing the eyes to move slowly towards the right. However, the left-sided vestibular system will continue to make the eyes move rapidly towards the left, and the brain will again attempt to move the eyes slowly towards the right for compensation. This cycle will happen repeatedly, leading to nystagmus. If the brain is unable to compensate, only the left-sided rapid movement will be observed. In any case, the fast component is the primary event. Therefore, by convention, the direction of nystagmus is determined by the rapid component, not the slow component.
@PeterJohnsАй бұрын
Yes, I've thought of that before. I would phrase the last sentence as "In vestibular neuritis, you see an abnormal HIT (meaning an poorly functioning VOR) while in dizzy strokes, the vestibular nerve is not usually affected and the HIT is normal (VOR is normal),
@worldaround6520Ай бұрын
@@PeterJohns thanks a lot for the video, sir.
@Clinylourembam Жыл бұрын
Thank you for making it clear regarding nystagmus
@mrcash98982 жыл бұрын
Great explanation. Greets from Brazil!
@johnkuo8552 жыл бұрын
What a great educational presentation. thank you so very much
@mkhatame862 жыл бұрын
As usual, an excellent explanation and review.
@greensky0110 ай бұрын
Thank you for this excellent explanation!
@Nishamukherjee199521 күн бұрын
thanks a lot sir for such a wonderful video.
@samstapes7471Ай бұрын
amazing explanation.
@mygtr20218 ай бұрын
Thanks PJ for sharing this infor and making this video. i hope i can help more people with this information and explanation. God Bless
@ritaszentgroti17088 ай бұрын
excellent video! thank you so much for making and sharing it!
@AlexanderRoux2 жыл бұрын
Educational AND soothing
@wikunda2 жыл бұрын
I am a physician from Thailand and I find your videos very informative and easy to understand. Thank you so much for making these series of video clip. I have one question for you, from your videos it seems like your patients can tolerate the examination very well, have you given them any medications prior to examination? From my own experience, it is really hard to perform the exam, even just to open their eyes to look for nystagmus. Thank you.
@PeterJohns2 жыл бұрын
Thanks for your kind words. I use medications if I need them. I'm guessing that's about 20% of BPPV, and about 50% or more of AVS.
@wikunda2 жыл бұрын
@@PeterJohns That's very quick response, thank you so much.
@NickPeitsch2 жыл бұрын
Actually, I get vertigo in all directions with vestibular neuritis depending on the head movements I perform. I also feel vertigo in my limbs (i.e. arms and legs spinning in opposite directions).
@PeterJohns2 жыл бұрын
If you have vestibular neuritis, you can have dizziness brought on by any head movements. However, the direction of the nystagmus is fixed to either the left or the right.
@alfredopampanga93562 жыл бұрын
Excellent, once again
@aawajnp2 жыл бұрын
thank you sir. excellent explanation as always.
@patrickbrewer14812 жыл бұрын
Thanks for the great content!! How long is the nystagmus typically present? Case question: Does the absence of nystagmus after the acute phase (more specifically, 10 days after onset of symptoms) and the presence of a positive head impulse test still point towards vestibular neuritis in the absence of any other signs or symptoms other than constant vertigo?
@PeterJohns2 жыл бұрын
Typically the nystagmus is pretty easy to see in the first couple of days. Some seem to lose it fairly early after that. It's important to remove fixation in some manner (like by asking the patient to look through a piece of paper) to bring out small amplitude nystagmus that can be missed if you ask the patient to "look at my finger". Yes, the head impulse test can be abnormal for quite some time after the spontaneous or gaze evoked nystagmus is gone. But by then they shouldn't have constant vertigo.
@patrickbrewer14812 жыл бұрын
@@PeterJohns fantastic. Thanks so much!
@patrickbrewer14812 жыл бұрын
@@PeterJohns how long is the typical time for symptoms to resolve?
@ArkDesignHD Жыл бұрын
Thank you very much, clear explanation
@Thankgodthereisnogod2 жыл бұрын
Thankyouu so much sir.... That's an amazing explanation....✨
@ericandrius2 жыл бұрын
Sr. can you make a video about treatment of vestiubular neuritis? I found myself happy but lost after the diagnosis of vestibular neuritis. Thank very much for your work
@PeterJohns2 жыл бұрын
That's a good idea.I'll put it on my list of things to make videos about.
@jessleen123410 ай бұрын
very well explained!!!
@Shantanu.Shandilya11 ай бұрын
Great video. Can you shed light on why Hypoactive lesions are mainly associated with Horizontal Nystagmus not Vertical? Thanks.
@PeterJohns11 ай бұрын
Thanks! And sure, in vestibular neuritis the most commonly affected part of the nerve is the superior branch. It supplies innervation of the horizontal canal and anterior canal. This produces the typical horizontal and torsional nystagmus. Much less commonly the inferior branch of the vestibular nerve is affected, and it supplies the posterior canal. This produces a vertical downward and torsional nystagmus. Sometimes both branches are affected. This paper explains it in more detail. www.neurology.org/doi/10.1212/wnl.0000000000003223
@Shantanu.Shandilya11 ай бұрын
@@PeterJohns Thank you. :)
@aminehafi-y1fАй бұрын
شكرا جزيلا تعلمت منك الكثير
@sergeyryabov72628 ай бұрын
Thanks for your job
@radvocado Жыл бұрын
Thanks Dr Johns! I just have a question about the first gentleman with nystagmus, I see his eyes are brown centrally and grey peripherally. Is that indicative of another condition? Thanks again for the great video!
@PeterJohns Жыл бұрын
Not that I'm aware of. I thought they look quite nice!
@Marco-sp2li Жыл бұрын
Hopefully you see this comment today… but when you say beating away or horizontal. Are you talking about the eyes twitching one way or the other?
@PeterJohns Жыл бұрын
Nystagmus has a fast component and a slow component usually. The direction of the nystagmus is defined by the faster component. Nystagmus can be vertical upwards or downward, horizontal or torsional. In vestibular neuritis it is horizontal/torsional and the fast component beats away from the affected ear.
@lucacortese2704 Жыл бұрын
This is gold Doc, thank you very much for sharing. I have a question for you: do you find good response to antiemetic drugs like Levosulpiride can be a useful criterion for distinguishing a peripheral vertigo vs a central one? Often in our ED in Italy we administer Levosulpiride to very symptomatic patients, who are difficult to examine at first. They almost always respond well. Then we proceed screening them with a detailed neurological exam and HINTS plus test. In borderline cases or if we are not that sure, can strong and almost complete response to medication be used as a further confirmation of a more likely peripheral cause than a central one? Thanks in advance
@PeterJohns Жыл бұрын
Short answer, no. Just as good response to an antacid to someone with chest discomfort cannot be relied upon as ruling out a cardiac cause for the chest pain, neither can the response to anti-emetic treatment be used to point the diagnosis away from a central cause. I'm not sure this has been studied, but of note, Tarnutzer's papaer in the CMAJ "Does my dizzy patient have a stroke" www.cmaj.ca/content/cmaj/183/9/E571.full.pdf does not address this idea in this rather comprehensive paper.
@lucacortese2704 Жыл бұрын
@@PeterJohns Thank you very much. I see your point. I was reasoning on the fact that seeing a patient enter our clinic with debilitating neurovegetative symptoms and strong difficulty walking, administering them the medication and then be able to fully examine them finding complete regression of gait problems and imbalance and collecting eumetric cerebellar tests could be a valid point towards vestibular neuritis rather than cerebellar stroke. It has never happened to me, but I assume in a cerebellar stroke it would be difficult to find complete regression of gait and imbalance symptoms with a drug like Levosulpiride that acts mainly on peripheral nervous system (and only at high concentrations on central nervous system). Not to mention the fact that in vestibular neuritis we have an inflamed but well alive nervous tissue, instead in cerebellar stroke we have nervous tissue that has started do die. Anyways, this is just my speculations, not confirmed or treated in any study, as you mentioned me. HINTS plus test is clearly a very much more valuable tool to rule out a cerebellar stroke. Thank you for your answer and interest in teaching and expanding the knowledge on the subject. My respects, Luca
@ihsanillahi1922 Жыл бұрын
Many thanks for the presentation.I am little confused about nystagmus in the lady at 1:19.Her nystagmus is beating towards the left ear and you mentioned she has right sided bppv. Am I missing something?
@PeterJohns Жыл бұрын
No, it's beating upwards vertically, and also torsionally towards her right ear. There is no nystagmus beating toward her left ear.
@dr.omarrashwan14 күн бұрын
Thank you❤❤❤❤❤
@raghibsohail880 Жыл бұрын
Sir i am suffering from vestibular disorder (tinnitus, hearing loss, vertigo). This problem was started 4 years before with ear drum reputured. But in last two year it got worsen. Currently i am facing same problem of nystagmus (why because i have left year surgery tympanoplacity before 8 months but after surgery things are same. From last two months my vertigo type has changed. My eyes are shaking right to left and left to right. I cant focus on things sometimes this problem solved in few minutes or something hours. But from yesterday i m facing this problem but not solved. My mind is working perfectly but i cant walk properly. Any solution or suggestions will be highly appreciated. I am taking betahistine 16 mg from 2 years.
@brunocera30742 ай бұрын
finally, thank you!!
@vanzandtVids Жыл бұрын
My 2.5 yr old is undergoing whole exome screening due to congenital nystagmus. I can’t seem to learn enough about what could be the cause. MRI normal. No onh. Peripherals blonde. 20/125 ☹️
@PeterJohns Жыл бұрын
Sorry, I know very little about congenital nystagmus.
@ggdefranca2 жыл бұрын
awesome job as usual! It seems that the fast phase of the nystagmus is away from the more rapid firing neural circuit, as it attempts to correct the visual fixation?
@PeterJohns2 жыл бұрын
Yes, it tries to correct the slow phase, which was drifting off the target.
@chih_yungkuo93112 жыл бұрын
Thanks!
@annaskrzypek10295 ай бұрын
could you please explain why peripheral nystagmus intensifies when looking in the direction of the fast phase/the healthy ear??
@PeterJohns5 ай бұрын
Look up Alexander's law. This video explains it in detail. kzbin.info/www/bejne/r37Tg2CMfMdobZo&ab_channel=Neuro-OphthalmologywithDr.AndrewG.Lee
@annaskrzypek10295 ай бұрын
@@PeterJohns i dont really understand it bc during the video the doctor says that it gets worse when looking in the direction of the affected ear yet when i search for alexander's law it states that it gets worse when looking in the direction of the fast phase which should be the healthy ear for a vestibular lesion or neuritis?
@annaskrzypek10295 ай бұрын
also i thought that alexander's law concerns only peripheral causes yet the doctor talks about lesions above the vestibular nucleus?
@PeterJohns5 ай бұрын
@@annaskrzypek1029 Try this one. kzbin.info/www/bejne/nWPYeHxrdt6Wopo&ab_channel=JoshuaKruger
@thephantasmagoricalperson41142 жыл бұрын
Could you explain why hearing loss is more a central thing than a peripheral one? Usually if there is someone with vertigo and hearing loss I would think there's a problem in the inner ear
@PeterJohns2 жыл бұрын
AICA strokes (Anterior Inferior Cerebellar Artery) infarcts part of the cerebellum as well as the labyrinth. So the patient has a cerebellar stroke as well as an acute loss of balance and a loss of hearing, as the cochlea is infarcted. So they will have an abnormal HIT, but also have an acute hearing loss. Now viral labyrinthitis can also present with AVS and hearing loss. Certainly looking for new hearing loss in patients with vertigo will increase the sensitivity, and decrease the specificity of HINTS for cerebellar stroke. What the incidence is of labyrinthitis vs an AICA stroke presenting with hearing loss, vertigo and abnormal HIT is not known. I would say this: If someone had a viral URI, developed ear pain and then tinnitus and/or hearing loss and vertigo, with no concerning features or risk factors for posterior circulation stroke, I'd probably call it viral labyrinthitis and send them home. If an older person or with stroke risk factors developed a sudden onset of vertigo and hearing loss at the same time, without URI or ear pain, and had an abnormal HIT, and a new hearing loss, I would work them up for AICA stroke. Every other patient in between these two scenarios is going to depend on your resources, your tolerance for risk, and local practices.
@thephantasmagoricalperson41142 жыл бұрын
@@PeterJohns thank you very much!
@chih_yungkuo93112 жыл бұрын
thanks a lot
@filipeporto60342 жыл бұрын
Awesome
@daniloasenov77952 жыл бұрын
Hi 👋, what is your opinion on intratympanic injections such as steroids and stem cells for treatment?
@PeterJohns2 жыл бұрын
For treatment of vestibular neuritis? I know of no studies related to that.
@ChaudharySomvirDalal2 жыл бұрын
I dont understand how to decide fast component and slow component
@PeterJohns2 жыл бұрын
The direction of the nystagmus which looks more like a "jerk" is the fast component. The slow component is the slower move back to the other side. Imaging falling asleep while sitting. The slow component is when you head starts to drop. The fast component is when your head is jerked back up.
@ChaudharySomvirDalal2 жыл бұрын
@@PeterJohns thank you very much .. you mean corrective saccadic jerk is fast component ?
@PeterJohns2 жыл бұрын
@@ChaudharySomvirDalal Of the nystagmus, yes. A saccade is the fast movement between two focal points.
@hananmrad77612 жыл бұрын
Does vestibular neuritis cause heavy head?
@PeterJohns2 жыл бұрын
I don't know that term.
@harishchandergoel51592 жыл бұрын
Nice
@bealis72 ай бұрын
My eyes tend to go up though :(
@jaymendoza84242 жыл бұрын
How is this visibuler nuritis treated??? I believe I have this please help?