USMLE Step 1 Autonomic Pharmacology | Active Recall Review

[@spencita]

YOU DESERVE AN AWARD. THIS ISNT JUST GREAT FOR USMLE. ITS GREAT FOR LIFE !!!!!!!!!!!!!!!!!!!!!!!!

[@kanishkam6020]

Admire your enthusiasm and teaching!

[@RamonCastillo-ku3kd]

Dr Rahul, you are a teacher for the world, continue your journey that is needed as never before.

[@hyguruprep]

Thank you so so much for your humbling feedback! Best wishes to you as well :)

[@rktpktpktrkt]

This was so helpful for me. I needed to get over the hurdle of autonomics because like you said, it ties into everything else. I appreciate this lecture so much!

[@collinosullivan3175]

Exceptional teacher thank you Dr. Damania

[@Dr.ibrahimaljarah]

It’s amazing You are very good teacher Thank you Dr Rahul

[@CyrenaDesiree]

This is sooo helpful. Thank you!!!

[@nurlandav6356]

You are the best. Thank you so much for your videos.

[@Sam_1964]

Outstanding teacher

[@sandilenkosi5745]

Thank you so much.!! Your webinars are amazing.!💯🙌🙌

[@hyguruprep]

Thank you so much!

[@ellaillustrates9350]

WONDERFUL!

[@hyguruprep]

thank you for watching!!

[@effatrafiuddin6026]

im loving your videos!!!

[@m3student663]

35:22, you made a mistake. Increased AV conduction wouldn’t have prompted atropine administration. Increased AV shortens the interval and leads to tachycardia. The reason behind atropine administration was bradycardia. The correct answer would be increased AV conduction delay or answer choice F, both would cause bradycardia.

[@hyguruprep]

Hi - thanks for the clarification! I may have meant to say increased AV nodal conduction delay. If you have increased AV nodal delay you have less ventricular response (i.e. ventricle does not contract as frequently due to decreased chronotropy & subsequently you have bradycardia on peripheral pulses). Atropine inhibits excess Ach at SA & AV node. Thanks for catching this question. Sorry about any inconvenience! Hope you enjoyed.

[@m3student663]

@@hyguruprep No problem, I enjoy reviewing concepts with your videos, they are great. Having listened again, you did say delay, so I’m guessing it might have just been a typo and got left out from the answer choice.

[@shreyatumu6088]

At 28:21 why can't the asnwer be neostigmine, isn't that also a ACHE inhibitor?

[@ramyoruppal8915]

Neostigmine is a quarternary amine, doesn't cross the blood brain barrier. won't help with the symptoms.

[@miltonbiswas9629]

Sir, I am 51yers old.at this age is it possible to pass and get residency?

[@demekeeligo64]

Amazing Dr

[@hyguruprep]

Thank you so much

[@sarahsalah3366]

tyyy this was great

[@vineethavakkapatla6873]

Incredible

[@michaelchidueme6266]

Wanted to clarify, wouldn't hydralazine be the hypertensive drug used in pregnant women that causes lupus? Love your videos! very helpful, thank you so much!

[@hyguruprep]

Hi yes! The most common medications associated with drug induced lupus are hydralazine, procainamide, isoniazid, methyldopa, chlorpromazine, quinidine, and minocycline. As the slide was going through sympathetic sympathomimetic modulators, I wanted to highlight methyldopa, however you're absolutely correct that hydral can also cause drug induce lupus! Watch for Anti Histone Ab

[@isaurareyesdejesus6188]

Thank you!

[@tariqtariq3208]

hi great video , i need a help , i get confuse when do we use adrenaline and when we use atrpine when person is brady cardic , can you briefly tell it , i mess both questions alot

[@yumnatimsaal3807]

atropine causes tachycardia so we give it if patient is bradycardic. Adrenalin is a bit tricky. In low amounts, it causes tachycardia (beta1) but in high amounts it causes bradycardia (alpha2). When we give it to a patient in anaphylatic shock, the patient has a depressed sympathetic response so giving this drug increases their vasoconstrictor abilities. I hope Dr. Damania can correct/elaborate on the points I have made! :)