High altitude physiology & maladaptation syndrome- HACE, HAPE and AMS | respiratory MBBS 1st year

High altitude respiratory physiology and maladaptation syndromes i.e acute mountain syndrome, high altitude cerebral edema and high altitude pulmonary edema
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[@debigdogk9563]

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Thanks for appreciation…

[@debigdogk9563]

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[@PhysiologyOpen]

My God…filled with your love

[@debigdogk9563]

I absolutely love ❤️ you and your teaching. You’ve taken some much time to explain very thing. God bless you ❤❤❤❤

[@PhysiologyOpen]

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[@lokeshkumarv6739]

Very clear.. Thanks

[@PhysiologyOpen]

Most welcome

[@user-pb6vu1hm2m]

Thank thank you soooooo much mam in my clg my physiology mam not explained she gone through slides I so wierd about how understad these all concepts of RS but u helped a lot mam once again thank you mam

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[@drvimalsinghgusain7927]

Excellent.

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[@user-gp8fo7do8j]

Superb explaination❤ mam... I need some key tips to understand like you and teach like you pls 😊

[@PhysiologyOpen]

Always connect things as much as you can…I try to find logic and why if everything …for that I can spend hours trying to search for answers

[@PhysiologyOpen]

Atleast start to think of why…you will generate very good questions, which is the key

[@army-st3dx]

Thanks

[@PhysiologyOpen]

Most welcome

[@ofabhay]

Mam please upload video on Heat Loss🙏

[@PhysiologyOpen]

Temperature regulation physiology mbbs 1st year kzbin.info/www/bejne/fKbbd3uabLOgisU

[@user-hp2xu3if6e]

I have a doubt mam. In HAPE condition, decreased PO2 causes vasoconstriction, but in high altitude adaptation in lungs there is a capillary recruitment phenomen which is opening up of capillaries and distension. These two statement are contrary and confusing me

[@PhysiologyOpen]

Capillary recruitment occurs as a passive phenomenon by stretching to accommodate increased output from right ventricle for adaptation. However decreased po2 leads to vasoconstriction…so they are two different mechanisms

[@user-hp2xu3if6e]

@@PhysiologyOpen thank you mam😊

[@PhysiologyOpen]

@user-hp2xu3if6e most welcome

[@army-st3dx]

I want to buy your lectures plz tell me how many lectures are there and how many time it will requires to complete it?

[@PhysiologyOpen]

If it’s for personal use…I will make lectures next year which will be uploaded on my app but it will take time probably an year

[@army-st3dx]

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[@PhysiologyOpen]

Yes

[@debigdogk9563]

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😇

[@Vasudeva061]

Maam i have small doubt here… while u are telling the pathophysiology of HACE, u told that vasodilation is leading to increased hydrostatic pressure and thats leading to edema …..yeah increasing hydrostatic pressures leads to push force and that causes edema ……my doubt is How come vasodilation lead to increased Hydrostatic pressure??? My point is hydrostatic pressure is nothing but the blood pressure at the capillary levels so vasodilation leads to decreased resistance(by increase in radius) and should decrease the hydrostatic pressure naa?? Are there any other dominant factors driving this or am thinking in a wrong way please explain maam… I m 3 rd year student carrying this doubt since 1st year😅

[@PhysiologyOpen]

Vasodilation of arterioles - will lead to increased pressure ahead of it (capillaries) but decreased arterial pressure behind it (peripheral resistance - resistance to flow)

[@Vasudeva061]

Okayy Thank u so much maam…. nothing gives satisfaction than understanding the concept 😊

[@debigdogk9563]

You forgot something…….👑👑👑

[@PhysiologyOpen]

🤗